From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.tamu.edu!news.utdallas.edu!news.starnet.net!wupost!kuhub.cc.ukans.edu!mbcf!walker Newsgroups: bionet.neuroscience Subject: Immunophenotyping brain cells - help Message-ID: <1995Nov1.165252.8936@mbcf> From: walker@mbcf.stjude.org Date: 1 Nov 95 16:52:52 -600 Organization: St. Jude Children's Research Hospital Lines: 23 We are immunophenotyping by flow cytometry normal mouse brain cells and find that while we can easily detect certain phenotypes in cell suspensions (either homogenized or following trypsin digestion), in situ detection (frozen or fixed) is very difficult. We are new to the brain and wonder whether others doing similar analyses have had problems of this nature and if so, is there a way around the problem. Any advice would be greatly appreciated - thanks. ========================================= William S. Walker, Ph.D. Department of Immunology St. Jude Children's Research Hospital Memphis, Tennessee USA 38105-2794 bill.walker@stjude.org Voice: (901) 495-2537 FAX: (901) 495-3107 ========================================= From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!prodigy.com!LEAA97A From: LEAA97A@prodigy.com (MR STEPHEN J ENOCH) Newsgroups: bionet.neuroscience Subject: need information on nystagmus Date: 2 Nov 1995 14:12:35 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 9 Sender: daemon@net.bio.net Distribution: world Message-ID: <013.04047825.LEAA97A@prodigy.com> NNTP-Posting-Host: net.bio.net could anyone send any information on labers congenital nystagmus. what i am looking for is any possible devices that help someone afflicted with this condition. also, do any health institutions exist that specialize with individuals who have this condition. i would appreciate any information anyone could provide regarding labers. thank you./ From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!EU.net!Germany.EU.net!Dortmund.Germany.EU.net!nntp.gmd.de!news.ruhr-uni-bochum.de!news.rz.ruhr-uni-bochum.de!jan From: jan@neuroinformatik.ruhr-uni-bochum.de (Jan Vorbrueggen) Newsgroups: bionet.neuroscience Subject: Re: visual illusion/pain? Date: 02 Nov 1995 12:34:03 GMT Organization: Institut fuer Neuroinformatik, Ruhr-Universitaet Bochum, Germany Lines: 15 Distribution: world Message-ID: References: <199511011212.NAA11200@orion.rz.mdc-berlin.de> NNTP-Posting-Host: vesta.neuroinformatik.ruhr-uni-bochum.de In-reply-to: rcb1@LEX.LCCC.EDU's message of 1 Nov 1995 06:13:37 -0800 In article rcb1@LEX.LCCC.EDU (Ron Blue) writes: Does anyone know how I can get a copy of a visual illusion that causes pain? My understanding is that it looks like a circle with vertical lines inside. "Paradoxical integration is illustrated with the Thunberg thermal grill illusion. [...] The illusion you're talking about is _not_ visual, but thermoreceptive. I can't imagine anything visual causing pain except an (unstructured) overdose of photons, which is detrimental to your health anyway. Jan From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!usenet.eel.ufl.edu!quapaw.astate.edu!quapaw.astate.edu!khurley From: khurley@quapaw.astate.edu (Kevin E. Hurley) Newsgroups: bionet.neuroscience Subject: CS-UR circuit in Aplsia Date: 2 Nov 1995 13:50:16 -0600 Organization: Arkansas State University Lines: 10 Message-ID: NNTP-Posting-Host: quapaw.astate.edu Keywords: neural-network, model, GENESIS I am constructing a computational model of the CS-UR circuit in Aplysia. I am using GENESIS 2.0 to create the simulation of classical conditioning. If anyone would like to correspond on this subject, please email me. If you have readliy available references, please send them. -- Kevin E. Hurley | Arkansas State University Major: Psychology | khurley@quapaw.astate.edu Minor: Computer Science | khurley@osage.astate.edu From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!pendragon.jsc.nasa.gov!news.msfc.nasa.gov!newsfeed.internetmci.com!chi-news.cic.net!news.math.psu.edu!psuvax1!news.eecs.nwu.edu!newsfeed.acns.nwu.edu!movietone.ils.nwu.edu!news.ecn.bgu.edu!vixen.cso.uiuc.edu!s.psych.uiuc.edu!kspencer From: kspencer@s.psych.uiuc.edu (Kevin Spencer) Newsgroups: bionet.neuroscience Subject: Re: Brainwaves frequency higher than Beta? Date: 2 Nov 1995 19:29:43 GMT Organization: UIUC Department of Psychology Lines: 17 Message-ID: <47b677$pnn@vixen.cso.uiuc.edu> References: <478oma$co@mn5.swip.net> NNTP-Posting-Host: s.psych.uiuc.edu X-Newsreader: NN version 6.5.0 #6 >In article <478oma$co@mn5.swip.net> m-16389@mailbox.swipnet.se (Danko) writes: > Is it possible to make the human brain > produce waves with frequency higher > than Beta (>30Hz)? Many neurons in the human brain fire at rates higher than 30 Hz. In the scalp-recorded EEG, however, most of the spectral power is in the below-20 Hz range, especially in the alpha (8-12 Hz) and delta (0-4 Hz) bands. ----------------------------------------------------------- Kevin Spencer Cognitive Psychophysiology Laboratory and Beckman Institute University of Illinois at Urbana-Champaign kspencer@p300.cpl.uiuc.edu / kspencer@psych.uiuc.edu ----------------------------------------------------------- From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!howland.reston.ans.net!news-e1a.megaweb.com!newstf01.news.aol.com!newsbf02.news.aol.com!not-for-mail From: seebury@aol.com (SEEBURY) Newsgroups: bionet.neuroscience Subject: Re: NMDA again Date: 2 Nov 1995 15:58:05 -0500 Organization: America Online, Inc. (1-800-827-6364) Lines: 2 Sender: root@newsbf02.news.aol.com Message-ID: <47bbct$m46@newsbf02.news.aol.com> References: Reply-To: seebury@aol.com (SEEBURY) NNTP-Posting-Host: newsbf02.mail.aol.com looking for BIOFEEDBACK sources for training seminars, equipment, and professional associations. From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!in1.uu.net!nctuccca.edu.tw!news.Edu.TW!newsserver.jvnc.net!news.caren.net!news.join.ad.jp!news.imnet.ad.jp!ripspost.aist.go.jp!ripspom!b0017 From: b0017@nibh.go.jp (Maurizio MORABITO; Tel.6661) Newsgroups: bionet.neuroscience Subject: Re: Brainwaves frequency higher than Beta? Date: 2 Nov 95 11:42:11 Organization: National Institue of Bioscience and Human-Technology(NIBH), Japan. Lines: 19 Message-ID: References: <478oma$co@mn5.swip.net> NNTP-Posting-Host: ripspom.aist.go.jp In-reply-to: m-16389@mailbox.swipnet.se's message of 1 Nov 1995 21:26:34 GMT In article <478oma$co@mn5.swip.net> m-16389@mailbox.swipnet.se (Danko) writes: Is it possible to make the human brain produce waves with frequency higher than Beta (>30Hz)? It's not uncommon to record 40Hz signals What effects would that have? Think about it -- Maurizio Morabito |"I for one could offer a lot of thoughts on any maurizio@nibh.go.jp| subject,but in many cases they would be based on | speculation at best, or misinformation at worst" Tsukuba, Japan | D.P.Chassin WWW = ftp://ripsport.aist.go.jp/pub/outgoing/maurizio/maurizio.html From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!NERO.GLIA.MDC-BERLIN.DE!BL From: BL@NERO.GLIA.MDC-BERLIN.DE ("Babis") Newsgroups: bionet.neuroscience Subject: Test Date: 2 Nov 1995 07:08:18 -0800 Organization: mdc Lines: 12 Sender: daemon@net.bio.net Distribution: world Message-ID: <4509211787@nero.glia.mdc-berlin.de> Test2 Charalampos Labrakakis Cellular Neurosciences Max-Delbrueck-Center for Molecular Medicine Robert-Roessle-Str. 10 13122 Berlin Germany phone: ++49 30 94062858 Fax : ++49 30 94063819 From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!pendragon.jsc.nasa.gov!news.msfc.nasa.gov!newsfeed.internetmci.com!tank.news.pipex.net!pipex!warwick!griffin.nott.ac.uk!pln1.life.nottingham.ac.uk!plxrlh From: plxrlh@pln1.life.nottingham.ac.uk (Richard Hughes) Newsgroups: bionet.neuroscience Subject: oocytes not expressing Date: Thu, 2 Nov 1995 14:01:45 UNDEFINED Organization: University of Nottingham Lines: 6 Message-ID: NNTP-Posting-Host: pplcsx.nottingham.ac.uk X-Newsreader: Trumpet for Windows [Version 1.0 Rev B] Does anyone know why RNA injected into oocytes sometimes gets translated , and sometimes not ! . Please answer if you have experience in expressing ligand gated receptors. Thank you in anticipation R.Hughes From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!usenet.eel.ufl.edu!tank.news.pipex.net!pipex!oleane!jussieu.fr!math.ohio-state.edu!uwm.edu!vixen.cso.uiuc.edu!usenet.ucs.indiana.edu!indyvax.iupui.edu!indyvax.iupui.edu!nntp Newsgroups: bionet.neuroscience Subject: Any golfers going to Neuroscience meeting??? Message-ID: <1995Nov2.073038.19739@indyvax.iupui.edu> From: "J. R. Simon" Date: 2 Nov 95 07:30:38 -0500 Nntp-Posting-Host: 134.68.153.15 Lines: 6 If you are a golfer and will be in San Diego for the Neuroscience meeting, drop me an email. Perhaps we could get away for a few hours and enjoy the local links. Thanks. Jay From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!LEX.LCCC.EDU!rcb1 From: rcb1@LEX.LCCC.EDU (Ron Blue) Newsgroups: bionet.neuroscience Subject: Re: Pain Inducing Patterns (fwd) Date: 2 Nov 1995 05:48:10 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 25 Sender: daemon@net.bio.net Distribution: world Message-ID: ---------- Forwarded message ---------- Date: Wed, 1 Nov 1995 16:00:53 -0500 From: Bob Babcock To: x011@lehigh.edu Subject: Re: Pain Inducing Patterns > Using your favorite paint program, start with a black background, and draw > a circle that almost fills the screen, preferably in at least a medium > resolution (640 x 480). Now fill the circle with alternating horizontal > bars of black and white, approximately 20 of each, and remove any > remaining outline of the circle. This was the simplest of a number of > examples shown in one of the neurology journals - I don't have the > specific reference but it might be in Pickover's book. > It sounds like you won't be impressed, but if this image triggered a > migraine or seizure, your mind might be opened. Perhaps "pain" isn't > quite the right word, but the context isn't exactly social angst either. I made a postscript file to these specs using GLE and viewed it with Ghostscript. The image seems quite innocuous. My office mate agreed, describing it as "the belly of a zebra viewed through a tube". I converted the image to a GIF file which can be viewed using the URL http://cfa-www.harvard.edu/~peprbv/pain.html  From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!CS.Arizona.EDU!news.Arizona.EDU!news.arizona.edu!bill From: bill@nsma.arizona.edu (Bill Skaggs) Newsgroups: bionet.neuroscience Subject: Re: LTP in hippocampus Date: 01 Nov 1995 22:25:44 GMT Organization: ARL Division of Neural Systems, Memory and Aging, University of Arizona Lines: 42 Distribution: world Message-ID: References: <476f65$7nt@sifon.cc.mcgill.ca> NNTP-Posting-Host: subiculum.nsma.arizona.edu In-reply-to: kevin@gaba's message of 01 Nov 1995 17:08:07 GMT kevin@gaba (Kevin Hellman) writes: > I have attached some citations... Although, I am not an > experienced physiologist, I am skeptical about the role ltp in > the hippocampus. The paradigms used require an unusual amount of > stimulation. LTP might be a form of learning, but is it really > the form of learning we use in our hippocampus? There is an explanation for the high level of stimulation required using the standard paradigm. The pathways that are commonly stimulated in LTP experiments evoke direct excitation, but they also excite interneurons and thereby evoke feedforward inhibition. The driving of interneurons is very fast, and much stronger than the driving of principal cells. Consequently, at low levels of stimulation, the feedforward inhibition of principal cells outweighs the direct excitation, and no LTP occurs. In many parts of the neocortex and hippocampus this occurs at ALL levels of stimulation, and no LTP occurs regardless of how large the stimulus is; but LTP can be obtained by pharmacologically blocking inhibition. In the pathways where LTP can be obtained without such measures, the level of feedforward inhibition saturates at a certain stimulus intensity, while the direct excitation keeps increasing. Eventually the excitation outweighs the inhibition, and enough postsynaptic depolarization occurs to permit LTP. This typically requires extremely strong stimulation, but only because the pattern of stimulation (many fibers activated all at once) is not the sort that the system is built to work with. It has now become clear that all that is really required for LTP to occur at a synapse is a burst of presynaptic activity in conjunction with strong postsynaptic depolarization. This occurs quite often in the naturally functioning hippocampus, but because of the feedforward inhibition it is difficult to bring about by artificial stimulation. In any case, there are now known to be ways of getting LTP that don't require such massive stimulation. One of them is the so-called "theta burst" paradigm, in which a single moderate stimulus is applied, then, after a 200 millisecond pause, a brief train of moderate stimuli. This works because the first pulse puts the inhibitory interneurons into a refractory state that persists for a few hundred milliseconds. -- Bill From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!info.ucla.edu!nnrp.info.ucla.edu!modem0-e.lifesci.ucla.edu!user From: stephan@psych.ucla.edu (Stephan Anagnostaras) Newsgroups: bionet.neuroscience Subject: Re: LTP in hippocampus Date: Thu, 02 Nov 1995 00:41:35 -0800 Organization: UCLA Lines: 25 Message-ID: References: <476f65$7nt@sifon.cc.mcgill.ca> NNTP-Posting-Host: modem0-e.lifesci.ucla.edu X-Newsreader: Yet Another NewsWatcher 2.0.2 In article , kevin@head.neurology.wisc.edu wrote: > > I was wondering if anyone can give me any sources or any information > > on LTP in the hippocampus. I just want to first, get an overview of such > > a phenomena, which I hope can then allow me to delve into the subject > > matter a bit more deeply. > > > > Thanking you in advance, > > > > John B. > > I have attached some citations... Although, I am not an experienced > physiologist, I am skeptical about the role ltp in the hippocampus. > The paradigms used require an unusual amount of stimulation. LTP Many paradigms do not require unusual stimuilation - in fact, it's very easy to get LTP in the hippocampus. I have no doubt that LTP is important in the hippocampus - the doubt arises as to what its role is in learning, etc. In any case, there are lots of normal stimulation paradigms, e.g., theta burst stimulation or paired stimulation - the latter barely requires much stimulation. -- STEPHAN ANAGNOSTARAS UCLA BEHAVIORAL NEUROSCIENCE STEPHAN@PSYCH.UCLA.EDU From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!LEX.LCCC.EDU!rcb1 From: rcb1@LEX.LCCC.EDU (Ron Blue) Newsgroups: bionet.neuroscience Subject: Re: visual illusion/pain? Date: 2 Nov 1995 11:01:51 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 15 Sender: daemon@net.bio.net Distribution: world Message-ID: References: NNTP-Posting-Host: net.bio.net On 2 Nov 1995, Jan Vorbrueggen wrote: > In article > rcb1@LEX.LCCC.EDU (Ron Blue) writes: > > Does anyone know how I can get a copy of a visual illusion that causes > pain? My understanding is that it looks like a circle with vertical lines > inside. I was able to get the picture at http://cfa-www.harvard.edu/~perprbv/pain.html I tried it out on 25 people. One out of six reported pain. Two were in a strong state of discomfort. Also most reported subjective color. Colors were blue, green, red, and orange. Ron Blue From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!tank.news.pipex.net!pipex!dispatch.news.demon.net!demon!mail2news.demon.co.uk!relay-4.mail.demon.net From: Pat Bermingham Newsgroups: bionet.neuroscience Subject: Re: Split-brain/consciousness Date: Thu, 02 Nov 1995 14:29:52 GMT Organization: None Lines: 50 Message-ID: <567550439wnr@adflex.demon.co.uk> References: <420gv8$763$1@mhadg.production.compuserve.com> <42f19n$5ca@southern.co.nz> Reply-To: pat@adflex.demon.co.uk X-NNTP-Posting-Host: relay-4.mail.demon.net X-Broken-Date: Thursday, Nov 02, 1995 14.29.52 X-Newsreader: Newswin Alpha 0.7 In article: <42f19n$5ca@southern.co.nz> bsandle@southern.co.nz (Brian Sandle) writes: > > Donna Tabish (102703.3346@CompuServe.COM) wrote: > : > : I would like to know if anyone has, is doing research on, is > : interested in, or knows internet locations of information > : concerning the possible correlation/similarities between the > : effect on consciousness/personality of the split-brain (either by > : surgery or in early developmental years) and the phenomena of > : dissociation/dissociated identity. > : > > Another thing which bears on the matter is that people can live quite > well and even be very intelligent with only a brain stem. WHAT????? WRONG. VERY WRONG. (Or was that a joke?) > Brian Sandle. Shell to snail? bsandle@southern.co.nz In answer to Donna's original question, look up Gazzaniga; Sperry; and Preilowski on medline or psychlit if you can. This is quite old stuff (60's - 70's). Sperry and co believed that in split brain patients "each hemisphere has its own private sensations, perceptions, thoughts, feelings and memories" and "they constitute two separate minds, two separate spheres of consciousness" This is however quite different from dissociated identity patients whose personalities can rank in the 30s or more. There is some suspicion that in many cases these "personalities" are created by reckless psychotherapists whose suggestions turn a minor disorder into this unlikely scenario, which they then believe they have "uncovered" in therapy, much like repressed memories. Other theories highlight the possibility of a self-hypnotic event during childhood. Either way, the situations are quite dissimilar. In the one case the two halves of the brain are severed from one another and the brain is thus hard-wired for dissociation. As far as I am aware, there is no evidence for any anatomical dissociation in multiple personality syndrome patients, and this might be considered a more "functional" problem. Hope this is of use. lys ------------------------------------------------------------------ Pat Bermingham, Adflex Ltd UK e-mail: patb@demon.co.uk ------------------------------------------------------------------ From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!UNF6.CIS.UNF.EDU!jander From: jander@UNF6.CIS.UNF.EDU ("John E. Anderson") Newsgroups: bionet.neuroscience Subject: Re: Long term potentiation Date: 2 Nov 1995 06:24:51 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 18 Sender: daemon@net.bio.net Distribution: world Message-ID: References: <491ka186@neubio.sld.ar> On 1 Nov 1995, Administrador del Nodo wrote: > In our own tradition we are hylozoist and so > see no need of searching for engrams; least to use these constructs > as Procrustean beds to provide function onto any experimental fact > that admits to be interpreted as bolstering LTP. What do you mean? > =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= > Prof. Mariela Szirko, > -- John E. Anderson, Ph.D. janderlu@msn.com jander@unf6.cis.unf.edu From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!LEX.LCCC.EDU!rcb1 From: rcb1@LEX.LCCC.EDU (Ron Blue) Newsgroups: bionet.neuroscience Subject: Re: Brainwaves frequency higher than Beta? Date: 2 Nov 1995 06:12:24 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 12 Sender: daemon@net.bio.net Distribution: world Message-ID: References: <478oma$co@mn5.swip.net> Yes, a strobe light will do it. My understanding is that if the frequency is right you will feel pleasure. Ron Blue On 1 Nov 1995, Danko wrote: > Is it possible to make the human brain > produce waves with frequency higher > than Beta (>30Hz)? What effects would > that have? > > >  From owner-neuroscience@net.bio.net Wed Nov 01 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!elroy.jpl.nasa.gov!lll-winken.llnl.gov!enews.sgi.com!decwrl!news-server.ncren.net!news.duke.edu!carswell From: carswell@acpub.duke.edu (Brett Carswell) Newsgroups: bionet.neuroscience Subject: Re: Web Cerebral Hypoxia & Ischemia Resources Page Date: 2 Nov 1995 18:55:18 GMT Organization: Duke University, Durham, NC, USA Lines: 23 Message-ID: <47b46m$bp6@news.duke.edu> References: <473u86$mkd@news.ox.ac.uk> NNTP-Posting-Host: carr6.acpub.duke.edu X-Newsreader: TIN [version 1.2 PL2] Mike D.R. Croning (michael.croning@st-cross.ox.ac.uk) wrote: : I am posting to inform the usenet community of a free web cerebral : hypoxia and ischemia resources page I have recently compiled. : The purpose of this world wide web page is to index links to brain : hypoxia and ischemia resources around the world - subjects currently : poorly covered by other neuroscience indexes. : The main focus of the page is to list primarily basic research going on : around the world by 3 categories: subject, department & individual : investigators. Clinical information will also be included and there is : a facility for researchers to submit their own links - by EMAIL. : Currently the page contains about 50 links to sites around the world. : WWW address:- : http://sable.ox.ac.uk/~scro0037/guide.htm : thanks : Mike D.R. Croning From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!lhc.nlm.nih.gov!crick.ser.bbnplanet.net!lamarck.sura.net!ra.nrl.navy.mil!news.math.psu.edu!CTCnet!newsfeed.pitt.edu!uunet!in1.uu.net!newsfeed.internetmci.com!info.ucla.edu!nnrp.info.ucla.edu!usenet From: Stephen Lessnick Newsgroups: bionet.neuroscience Subject: cortical-spinal conections Date: 3 Nov 1995 05:49:51 GMT Organization: UCLA Lines: 13 Message-ID: <47cahv$fit@saba.info.ucla.edu> NNTP-Posting-Host: ts21-10.wla.ts.ucla.edu Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.1N (Windows; I; 16bit) To: all I have recently been asked to find info on the following, but have not been particularly successful. Does anyone have information regarding: 1) Why (from a teleological {sp?} point of view) does the right hemisphere control (and receive information predominantly from) the left side of the body and visa-versa? I'm sure there have been a number of theories advanced. If you have references for the theories it would be really helpful. 2) When in evolution does this pattern of crossed connections arise? Thanks! From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!cs.utexas.edu!news.sprintlink.net!wizard.pn.com!Germany.EU.net!EU.net!newsfeed.internetmci.com!info.ucla.edu!nnrp.info.ucla.edu!usenet From: Stephen Lessnick Newsgroups: bionet.neuroscience Subject: cortical-spinal conections Date: 3 Nov 1995 05:50:34 GMT Organization: UCLA Lines: 13 Message-ID: <47caja$fit@saba.info.ucla.edu> NNTP-Posting-Host: ts21-10.wla.ts.ucla.edu Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.1N (Windows; I; 16bit) To: news:bionet.neuroscience I have recently been asked to find info on the following, but have not been particularly successful. Does anyone have information regarding: 1) Why (from a teleological {sp?} point of view) does the right hemisphere control (and receive information predominantly from) the left side of the body and visa-versa? I'm sure there have been a number of theories advanced. If you have references for the theories it would be really helpful. 2) When in evolution does this pattern of crossed connections arise? Thanks! From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!LEX.LCCC.EDU!rcb1 From: rcb1@LEX.LCCC.EDU (Ron Blue) Newsgroups: bionet.neuroscience Subject: Re: Split-brain/consciousness Date: 2 Nov 1995 11:42:28 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 43 Sender: daemon@net.bio.net Distribution: world Message-ID: References: <567550439wnr@adflex.demon.co.uk> > > Donna Tabish (102703.3346@CompuServe.COM) wrote: > > : I would like to know if anyone has, is doing research on, is > > : interested in, or knows internet locations of information > > : concerning the possible correlation/similarities between the > > : effect on consciousness/personality of the split-brain (either by > > : surgery or in early developmental years) and the phenomena of > > : dissociation/dissociated identity. >>>>>CUT>>> > Either way, the situations are quite dissimilar. In the one case the two > halves of the brain are severed from one another and the brain is thus > hard-wired for dissociation. As far as I am aware, there is no evidence for > any anatomical dissociation in multiple personality syndrome patients, and > this might be considered a more "functional" problem. I disagree. Oliver Sachs reported that one of his patients while having a migraine aura had the separation of self into two consciousness. This can be understood as the self as an attractor of all experiences. When the migraine created choas two selves were created. When the client tried to integrate them back into each other they could not. It sort like a disk balanced on a stick. When it is sent viberating wildly. Two centers emerge oscillating around the really center. Further support was collected with vestibular stimulation. A woman who was paralyzed in her arm due to brain damage (Science News, October 21, 1995 page 271) was NOT conscious of her condition. When cold water was put in her ear she now knew she was paralyzed. When the effect wore off she now knew she was NOT paralyzed even though she was. She denied ever saying she was paralyzed and did not remember the events. In Nature 31 August 1995 page 778 researcher G. Bottini, E. Paulesu, R. Sterzl, E. Warburton, R.J.S. Wise, G. Vallar, R.S.J. Frackowlak, & C. D. Frith in their article "Modulation of conscious experience by peripheral sensory stimuli" revealed that cold water ran in the ear of brain damaged patient who had lost the sense of touch regain the sense temporally. "We show that in normal subjects touch and vestibular signals share projectionss to the putamen, insula, somatosensory area II, premotor cortex and supramarginal gyrus. In our patient a subset of theres regions (right putamen and insula) was pared by the lesion and was maximally active when touch and vestibular stimulations were combined." (Bottini & etc. 1995) All these example suggest correlational opponent processing to me. Ron Blue From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!news.sprintlink.net!ns1.win.net!bugs From: Joseph Campellone Newsgroups: bionet.neuroscience Subject: Re: guillain-barre syndrome & nerve growth Date: 3 Nov 1995 00:15:29 GMT Organization: Win.Net Communications, Inc. Lines: 3 Message-ID: <47bmv1$9i7@ns1.win.net> References: <472nn1$omm@netnews.upenn.edu> NNTP-Posting-Host: dial-41.win.net Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.1N (Windows; I; 16bit) If you are at the Univ of Penn, perhaps you should try to see Dr Art Asbury, he has probably published more on GBS than anyone... From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!info.ucla.edu!library.ucla.edu!nnrp.info.ucla.edu!usenet From: Stephen Lessnick Newsgroups: bionet.neuroscience Subject: cortical-spinal conections Date: 3 Nov 1995 05:51:33 GMT Organization: UCLA Lines: 13 Message-ID: <47cal5$fit@saba.info.ucla.edu> NNTP-Posting-Host: ts21-10.wla.ts.ucla.edu Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.1N (Windows; I; 16bit) I have recently been asked to find info on the following, but have not been particularly successful. Does anyone have information regarding: 1) Why (from a teleological {sp?} point of view) does the right hemisphere control (and receive information predominantly from) the left side of the body and visa-versa? I'm sure there have been a number of theories advanced. If you have references for the theories it would be really helpful. 2) When in evolution does this pattern of crossed connections arise? Thanks! From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!tank.news.pipex.net!pipex!lade.news.pipex.net!pipex!news00.sunet.se!sunic!mn6.swip.net!news99.sunet.se!news.funet.fi!ousrvr3.oulu.fi!jheikkil From: jheikkil@cc.oulu.fi (Juhani Heikkila) Newsgroups: bionet.neuroscience Subject: Workshop - Brain Receptor Studies in vivo Date: 3 Nov 1995 10:08:32 GMT Organization: University of Oulu, Computer Services Center Lines: 48 Message-ID: <47cpn0$4du@ousrvr3.oulu.fi> NNTP-Posting-Host: sun2.oulu.fi Mime-Version: 1.0 Content-Type: text/plain; charset=ISO-8859-1 Content-Transfer-Encoding: 8bit X-Newsreader: TIN [version 1.2 PL2] WORKSHOP BRAIN RECEPTOR STUDIES IN VIVO Date: December 8.-9.12.1995 Place: Oulu University, Faculty of Medicine, Main auditorium, Kajaanintie 52 A This workshop deals with current development of brain receptor studies including radiochemistry, modelling, new developments in PET and SPET devices, neuropsycho-pharmacology, current and future use of brain receptor studies in neuropsychiatric disorders in adults and children. We focus our attention on beta-CIT tracers and have succeeded in having the most prominent speakers at this field as speakers (H.J. Biersack, K.Bergstroem, Th.Brucke, C.Halldin, J.Hietala, J.Kuikka, B.Maziere, J.L.Neumeyer, J.Tiihonen). Present and future improvements in PET and SPET devices will be presented by S.Larsson. Commercial exhibition will be organized at the same place and companies are offered opportunity to short presentations dealing with new developments in psycho- and radiopharmaceuticals and imaging devices. Organisers: Div. of Nuclear Medicine and Laboratory of Oulu University Hospital Depts. of Clin. Chem., Psychiatry and Neurology, University of Oulu Finnish Academy Finnish Society of Psychiatry Finnish Society of Nuclear Medicine This workshop belongs to Finnish National Research program and to continuing education program of the European School of Nuclear Medicine Further information: Assist. professor Aapo Ahonen Physicists Juhani Heikkila phone: 358-81-3154444 or or Pentti Torniainen M.D. Olli Puronto -3154443 phone: 358-81-3154453 FAX: 358-81-3155541 Div. of Nuclear Medicine Dept. of Clinical Chemistry FIN-90220 Oulu, Finland e-mail: juhani.heikkila@oulu.fi www: http://koivu.oulu.fi/~ptornia/receptor.html Registrations: Secretary Anne Ollila Address: Dept. of Clinical Chemistry, FIN-90220 Oulu, Finland FAX: 358-81-3154451 Deadline for registration: 27.11.1995 Registration fee is 600 FIM (about 200 DEM, 130 USD free of charge) Bank account: 'Brain receptor studies in vivo' OP-Oulu 574089-236081 E-mail registrations: juhani.heikkila@oulu.fi From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!in2.uu.net!hearst.acc.Virginia.EDU!newslink.runet.edu!dmcclain From: dmcclain@runet.edu (Dennis McClain-Furmanski) Newsgroups: bionet.neuroscience Subject: Re: Location of language processing Date: 3 Nov 1995 08:41:28 GMT Organization: Radford University Lines: 563 Distribution: world Message-ID: <47ckjo$2ga@newslink.runet.edu> References: <4744gu$n0s@epervier.CC.UMontreal.CA> NNTP-Posting-Host: james-gw.runet.edu X-Newsreader: TIN [version 1.2 PL2] belanm@tornade.ere.umontreal.ca wrote: : I would like to know if any study has been done to determine where : the center of language processing is located when deaf persons are : concerned. And if so, is it differently located than in ther non-deaf. : Marc A. Belanger. Here's some relevant material. It may not directly answer, but might lead you to some answers via references. Remember that 'left' and 'right' are usually just majority cases. Some people are reversed and some are less differentiated than others. The disclaimer comes first: this was an undergrad project. It is neither current nor necessarily very accurate. It was a rhetoric project more than a scientific one, though I tried to cover both as best as I could at the time. Abstract Sign languages, like many other languages, have been studied in terms if their components. After they have been broken down to their basic units, they can be described and compared. When these descriptions are based on the components of the languages in question, they are sufficient to describe. However, unless the descriptions used are sufficient to describe both of the languages being compared, they will not serve as adequate for comparison. This paper will attempt to provide an initial deconstruction of a communication paradigm which will be shown to be faulty in its construction due to its failure to adequately comply with the empirical evidence on which it depends for description, and to provide both a scientific and cultural rationale for this deconstruction. Introduction Spoken langauge is frequently studied in terms of morphemes, the basic unit of meaning. Likewise, sign languages are studied in their components, called "cheremes" (Stokoe, 1972). These basic units are "tab", "sig" and "dez", corresponding to location of the sign, configuration of the hands, and movement of the hands. Yet these are not enough to cover all the nuances of some signs. As the author of the above convention states, "a sentence, S, in American Sign Language may be seen to have two sensible components which are both vehicles for its intelligible import ... the facial, F, and the manual, M." (Stokoe, 1972). Facial representation has been added to notation created to relate the above concepts, yet it too fails in some cases. It is perhaps a case of unintentional chauvinism on the part of speaking linguists, who attempt to describe something based on the concepts created from their own experience. Unfortunately, there are components of sign language which elude any description in terms of spoken language, because they are inherently impossible to accomplish, or at best, matters of evolution of the language and better studied as etymology. Spoken Language As Serial Communication All spoken langauge shares a characteristic which is familiar to the study of cognitive science; it is serial. That is to say, it occurs with its individual components happening sequentially. Specifically, it occurs in receiving in the order of (1) auditory signal comprehension, (2) phoneme identification, (3) auditory word identification, and (4) cognition, which includes the application of semantic rules (Gordon, 1990), and in the reverse in the production of speech, ending with signal production instead of comprehension. Since it is physically impossible to say more than one thing at the same time, this seems an almost ridiculous observation to make. Yet, if we are to compare sign and spoken langauge, this concept must be addressed. Parallel Channels Sign language, being a visual language and involving up to the entire body of the signer, can often contain components which occur simultaneously. In the computer metaphor used so often by cognitive science, this is said to be parallel. Even if the components of the particular message being transferred arrive in a serial fashion, as long as the syntactic construction of the message follows the general syntactic construction rules of a non-serial based language, a linear view used to explain the message will be inaccurate (Goodall, 1987). Unless this specific difference is taken into account, the traditional method of description as adapted to sign language will fail. This simultaneity manifests itself in various ways. The first and most obvious is a restatement of Stokoe's earlier observation. There are more than one physical component to signs, and these must be recorded and analyzed in the context of their common occurrence. Second, following Stokoe's second comment, the sign consists of more than the manual components, including the face and various other elements of kinesics. Third, and as far as I have been able to determine almost unaddressed, is the simultaneous occurrence of more than one otherwise independent sign to form a compound sign. The sole instance of publication which I have been able to find on this version of simultaneity in communication has been a series of observations of Japanese Sign Language, although the nature of the usages these observations cover are identical to many instances of terms I am personally familiar with in ASL. The author of this work goes as far as to label the use of the word "simultaneity" in this instance to be a "cognitive" concept, rather than one of physical coincidence. (Peng, 1978). As an example, the ASL sign for "standing" -- two fingers extended downward onto the palm of the other hand, and the sign for "not paying attention", head tilted to one side, pupils turned to the corner of the eye tilted more upwards, and the tongue sticking out the corner of the mouth tilted more downwards, represents the clause "standing around not paying attention." This sign should be differentiated from the series of two concepts presented by the English "standing around and not paying attention" which indicates two related but separate concepts. If the intended meaning had been that the person was standing around, and while doing so was not paying attention, the signs would be presented in sequence. Instead, the condition of standing around obliviously, as a single statement of behavior, is presented. The tendency towards simultaneous signing is responsible for the creation of new signs from two or more old ones. This pattern of "blending" (Peng, 1978) is the source of such combined signs as the familiar (as opposed to the intimate) "I love you", created out of combined manual alphabet signs for "I", "L" and "Y". It is also the root of single signs with origins in two or more signs such as "teacher", being both hands palm down with thumbs tucked below, and brought down in an arc in front of the face, as opposed to the now more formal individual signs for teach (the hands brought directly out from the face) followed with the "agent" sign (hands directly in front of the elbows, palms towards each other, and sliced downwards in parallel). This blending of signs is so common, and considered so much a part of the language of sign, that the compound signs "apple+drink" for apple juice was one of the instances used to support the hypothesis of animal-acquired language, as quoted by Patterson in her paper on the language development in the gorilla Koko. (Patterson, 1978). Parallelism in Signs It would hardly be prudent to consider a change, addition or deletion of a theoretical framework without showing how it might be of some use. The consideration of the parallel information being produced, transmitted, received and processed, as multiple channels of simultaneous and mutually supporting information, can be used to explain such phenomena as the syntactic structure of American Sign Language. In ASL, it is just as proper to sign "my + book" as it is to sign "book + my". Similar arrangements of noun and verb pairs, subject and object ordering, and clauses within sentences are so frequent that a common explanation for this is that ASL has little or no set syntactic ordering of its components, (Wilbur, 1987), and some have even gone as far as to say that ASL has developed "avoidance strategies to compensate for constrained syntactic rules", (Freidman, 1976) almost as though the structure of ASL were somehow cheating in the production of language as compared to the rigidity of English. While variable syntax is a suitable explanation when only considering the language from a serial-oriented viewpoint, it does not address how such arrangements can be considered equivalent in semantic content. Wilbur (1987) addresses this by using the concept of "iconic representation", which is described as "a reflection in language of the actual state of affairs in the real world". If one accepts that the information transmitted in sign language resides in more than one channel, then it is not a large step to accept that operating in a real world with multiple channels of communication leads one's mind to think in representations of that information as they occur, that is, as parallel pieces to a single puzzle. Rather than being a work-around for a highly structured (and questionably considered 'standard') spoken langauge, the variable syntax of sign language instead represents the nature of the method of processing the information, which in terms of cognitive science is parallel processing of information received in parallel channels, even though the language is sometimes constrained to serialized or segmented, sequential production due to the limited amount of information which can be conveyed with each individual sign. This has led at least one researcher to apply the concepts of "relational grammar" to the study of ASL (Padden, 1988), as being composed of "primitives", or grammatical relationships which are not expressed as word or clause order, but instead create the rules where by word and clause order are governed. This research indicates that there is indeed ordering in sign language, but it is as often the result of the easiest transition between different signs as any semantic consideration, yet it carries the same information content. I personally feel it to be quite a stretch of the intent of the term grammar to try to include the convenience of physical motion as a governing dimension in communication within that term. Lateralization and Specialization In order to promote a parallel view of sign communication, it is necessary to differentiate and justify this from the serial view presented by previous empirical investigation. In support of his theory of syntax, Chomsky (1965) stated that "It may well be that the general features of langauge structure reflect, not so much one's experience, but rather the general character of one's capacity to acquire knowledge." The sequential production fo spoken language outlined above corresponds well with the sequence of neuroanatomical structures involved in production of spoken language, specifically the hippocampal and cortical memory areas, Wernicke's area, Broca's area, and the motor cortex (Kalat, 1992). If it can be taken that the brain structures involved, in less formal terms, the hard wiring is indeed responsible for the structure of the language, then one would expect that a visually oriented language would make use of equivalent structures within the visual area. In fact there are fundamental differences in the portions of the brain used for visual comprehension as opposed to verbal (the corresponding motor activity being controlled in all cases by the motor cortex). Friedrich (1990) observes that the left hemisphere, which controls the primary verbal areas, operate in an analytic and sequential manner, whereas the right hemisphere, which controls the spatial and visual recognition functions, operates primarily in a holistic and simultaneous manner. In the case of deaf communication, it has been shown (McKeever, Hoemann, Florian & VanDeventer, 1976) that the cerebral functions governing communication in deaf persons is not the same as those in hearing persons. More specifically, Ross, Pergament and Anisfeld (1979) showed that the right hemisphere of deaf persons was specialized for sign recognition, and was used in exactly the same fashion with very similar results as the left hemisphere of hearing persons was specialized for word recognition. If those areas of the right hemisphere used in the production and comprehension of sign language were fundamentally different from the corresponding left hemisphere speech areas, we would expect to find that the functioning within those areas to differ also. This is in fact the case. As Shand and Kilma (1981) show the differences in processing of sign language are due in part to differences in sensory storage, and also differences in processing of "static vs. changing-state" input, but even these are not sufficient to cover all the differences. These differences cannot be attributed to sensory-motor memory, according to Liben, Nowell and Posnansky (1978), as the memory of signs in deaf people is not organized according to the physical determinants of the sign components. Bellugi, Kilma and Siple (1975) further show that the memory storage of signs in deaf persons is organized according to "simultaneous formational parameters" which when taken as individual components are found to be arbitrary and therefore void of inherent meaning. It is this existence of meaning in simultaneous components which individually are meaningless which point solidly at a holistic basis for considering the meaning in the signs, and a multiple parallel channel paradigm for describing their transfer. Cognitive Computational Model Although the mechanistic view posited by cognitive science, examining the mind as a system of functional components integrated for the purpose of computation, is somewhat reductionist, and therefore less than ideal for investigation of holistic concepts such as the operation of the right hemisphere, it does lend itself well to creation of some fairly inclusive models of mental performance. In fact, the design of neural networks as programs for parallel processing computers, those computer systems intended to mimic some operations of the human brain, are a primary result of successful application of the theory to technology (Willis, Montague, Morris & Tham, 1993). Previous computer designs were all basically serial in nature; they acted on data and instructions in a linear fashion. Although these systems were capable of processing the information required to produce a realistic looking image, the time frame required was too long to allow sequential pictures to be shown to give any sense of motion. Lately, computers with several processors working in parallel have been used to more efficiently portray three dimensional images. Only these parallel devices are able to handle the enormous mass of data fast enough to display movement in the image in anything approaching real time equivalency. Justifying the concept of parallel processing in the brain by drawing an equivalent from computer science would seem quite a stretch. However, the flow of concepts runs in the opposite direction in this case. According to W. Daniel Hillis (1987), designer of the massively parallel computer The Connection Machine, his blueprint for the design of his machine was the human mind, due entirely to the fact that it uses the principle of parallel processing to comprehend an entire image all at once. It is able to do so due to the enormous number of interconnections between neurons, even though the neurons themselves are thousands of times slower than the silicon gates in computer chips. The parallel processing of image information proceeds even prior to reaching the brain. The neurons of the retina are interconnected in such a way as to respond to aggregate stimuli such as an edge as a single non-point entity (Hayward & Varela, 1992). In comprehending the complete sign, all aspects of its production must be considered. In the cognitive terms of Lieser and Gilleron (1990), in order to perceive the brain must calculate simultaneously any impinging aspects of space, time and length. Each of these may contain any number of components, including continuous spectrums of measurement as opposed to discrete entities. While a linear or serial computational model may be used to calculate each of these given enough time, the requirement of simultaneity of processing in order to construct a conscious image of a three dimensional moving figure such as a signer precludes serial processing as a possibility. While this would seem to serve as the necessary basis for describing visual imagery, Rollins (1989) asserts that these same computational structures are required for the production of language of any sort. His components to language which act as functional equivalents to space, time and length are semantics, process and syntax. From this it seems that not only is the cognitive computational model useful in the understanding of visual modes of communication and specifically sign langauge, but quite possibly in understanding language in general. Science and Social Responsibility A society which supports science should expect that science to in turn act responsibly towards that society. In the case of the investigation of sign language, this becomes apparent when we consider that the particular views adopted by science can have a significant effect on how society behaves towards the deaf segment of that society. There has been considerable bias towards the deaf in the past, and quite often it is based on the view that they are deficient in communicative ability. This opinion is fed significantly if the consideration of the people is based on their mode of communication, and this in turn is considered on the basis of an ineffective cognitive model which wrongfully labels these people as deficient. If one insists on using the serial processing and transmission model of communication when considering the parallel processing and transmission of the deaf, one can end up with results similar to those of Bishop (1983) which are based on an "inherent grammatical problem" made visible by basing the test on rigid ordering of response in verbal testing. This is obviously a flawed concept if one participates in a mode of communication where grammatic ordering is irrelevant. Results such as this have been used to justify the concept of disability in the deaf, even though testing has shown that deafness does not correspond to cognitive deficit (Lillo-Martin, Hanson & Smith, 1992). Even the idea that sign language is a substitute for "real" language should be questioned when findings show that it is learned faster that spoken language (Orlansky & Bonvillian, 1985). Anthropological investigation has shown that it is likely that sign language was in fact the first language used by humans (Hewes, 1973; Teodorsson, 1980). When one investigates the more useful language concepts such as creativity, then one finds results such as those from Marscharck and West (1985) which indicate that basing language testing on English-like concepts seriously underestimates the cognitive and linguistic abilities of the deaf. Even Geoffery Coulter (1993), editor of Phonetics and Phonology: Current Issues in ASL Phonology, questions the validity of using the concept of syntactic comparisons in investigating sign language, due to the unique nature of sign language. If one follows the theoretical model that the mode of communication used allows one preferential access to the hemisphere which processes it, then one can arrive at the conclusion that use of sign language would provide for better access to the right hemisphere and its holistic computational operation. This is in fact suggested by Hendren (1989); sign language is suggested as a communicative tool for all students for just this type of thinking. A significant point for its inclusion into eduction is made by the observation that if a person learns to sign first, and speak later, then that person is able to communicate in both ways at once, with the speech following the normal rules of grammar, and the sign following its normal rules flexible ordering, without conflict of confusion between the two (Jones & Quigley, 1979). This goes beyond the concept of bilingualism, and offers people a chance to communicate from both hemispheres at once, creating clearer and more meaningful communication. Given the above, it is understandable that the assumed supremacy of spoken language as the standard by which communication is measured has come into question (Televik, 1981). This should be reflected in the way that deaf people are viewed by society. Charrow and Wilbur (1975) suggest that rather than disabled, it would be more appropriate to consider the deaf as merely the third largest non-English speaking minority in the U.S., and that they should be treated in the respect of a cultural minority rather than a communicatively disabled one. This seems to be preferential to the existing view which results in society trying to force English speaking concepts on these people. Such efforts are doomed to failure (Grove, O'Sullivan & Rodda, 1979), and actually result in damage to the individual (Stokoe, 1978) by alienating them from their native culture, which is inseparable from their language (Stevens, 1980). Conclusion Although science must proceed as a lowest common denominator in its descriptions of the real world, and as such requires a language of its own which communicates all of the various parts of the world which it attempts to describe, it also is responsible to be the clearest form of communication. While those theoretical concepts produced based on serial communications are adequate for many purposes, and understandable by most people, they are not adequate to describe communications which occur in a fashion outside the realm of experience of their backgrounds, i.e. parallel forms of communication such as sign langauge. As stated by Lieberman (1984) "the initial functional value of syntax may rest in the limitation of semantic interpretation." As long as the semantic interpretation of a language is based on concepts found to be too limited for a complete understanding, the resulting observations of syntactic structure will be doomed to failure as proper descriptions of the reality they portray. In order to describe sign language properly, and particularly to compare it with spoken languages, it is necessary to use a theoretical framework which takes into accurate account the natures of the languages. Concepts derived from serial based languages are insufficient to describe parallel based languages, and are therefore insufficient for comparisons. When social factors regarding the results of one paradigm show obvious bias and damage, and a competing paradigm appears to reduce or nullify the negative effects, while providing for better communication for all as well as better description of reality, I think it imperative for science and society either accept the latter, or provide for a yet greater improvement in science while still reducing the bias created by the outdated paradigm. REFERENCES Bellugi, U., Klima, E. & Siple, P. (1975). Remembering in Signs. Cognition, 3, 93-125. Bishop, D. V. (1983). Comprehension of english syntax by profoundly deaf children. Journal of Child Psychology and Psychiatry and the Allied Disciplines, 24, 415-434. Charrow, V. & Wilbur, R. B. (1975). The deaf child as a linguistic minority. Theory Into Practice, 14, 353-359. Chomsky, N. (1965). Aspects of the theory of syntax (p. 180). Cambridge, MA: MIT Press. Coulter, G. and Anderson, S. (1993). Introduction. In Geoffery Coulter (Ed.) Phonetics and phonology, Volume 3: Current issues in ASL phonology. San Diego, CA: Academic Press. Friedman, L. (1976). Phonology of a soundless language: Phonological structure of American Sign Language. Doctoral dissertation, University of California, Berkeley. Friedrich, F. J. (1990). Frameworks for the study of Human Spatial Impairments. In R. P. Kesner & D. S. Olton (Eds.), Neurobiology of comparative cognition (pp 317-338). Hillsdale, NJ: Lawrence Erlbaum Associates. Goodall, G. (1987). Parallel structures in syntax (p. 172). Cambridge, UK: University Press. Gordon B. (1990). Human language. In R. P. Kesner & D. S. Olton (Eds.), Neurobiology of comparative cognition (pp 21-50). Hillsdale, NJ: Lawrence Erlbaum Associates. Grove, C., O'Sullivan, F. D. & Rodda, M. (1979). Communication and language in severely deaf adolescents. British Journal of Psychology, 70, 531-540. Hayward, J. W. & Varela, F. J. (1992). Gentle Bridges (p. 59). Boston: Shambala. Hendren, G. R. (1989). Using sign language to access right brain communication: A tool for teachers. Journal of Creative Behavior, 23, 116-120. Hewes, G. W. (1973). Primate communication and the gestural origin of language. Current Anthropology, 14, 5-24. Hillis, W. D. (1987, June). The connection machine. Scientific American, pp. 174-182. Jones, M. L., & Quigley, S. (1979). The acquisition of question formation in spoken english and american sign language. Journal of Speech and Hearing Disorders, 44, 196-208. Kalat, J. W. (1992) Physiological Psychology (p. 175). Pacific Grove, CA: Brooks/Cole. Liben, L. S., Nowell, R. C. & Posansky, C. J. (1978). Semantic and formational clustering in deaf and hearing subjects' free recall of signs. Memory and Cognition, 6, 599-606. Lieberman, P. (1984). The biology and evolution of a language. Cambridge, MA: Harvard. Lieser, D. & Gillieron, C. (1990). Cognitive science and genetic epistemology (p. 25). New York: Plenum. Lillo-Martin, D. C., Hanson, V. L. & Smith, S. T. (1992). Deaf readers' comprehension of relative clause structure. Applied Psycholinguistics, 13, 13-30. Marscharck, M. & West, S. (1985). Creative language abilities in deaf children. Journal of Speech and hearing Research, 28, 73-78. McKeever, W. F., Hoemann, H. W., Florian, V. A. & VanDeventer, A. D. (1976). Evidence of minimal cerebral asymmetries for the processing of english words and american sign language in the congenitally deaf. Neurophysiologia, 14, 413-423. Orlansky, M. & Bonvillian, J. (1985). Sign language acquisition: Language development in children of deaf parents and implications for other populations. Merrill-Palmer Quarterly, 31, 127-143. Padden, C. (1988). Interaction of morphology and syntax in American Sign Language. New York: Garland. Patterson, F. (1978). Linguistic capabilities of a lowland gorilla. In F. C. C. Peng (Ed.) Sign language and language acquisition in man and ape. (pp. 161-202). Boulder, CO: Westview Press. Peng, F. (1978). Introduction. In F. Peng (Ed.) Sign language and language acquisition in man and ape. Boulder, CO: Westview Press. Rollins, M. (1989). Mental imagery (p. xv). New Haven, CN: Yale. Ross, P., Pergament, L. & Anisfeld, M. (1979). Cerebral lateraliztion of deaf and hearing individuals for the linguistic comparison judgements. Brain and Language, 8, 69-80. Shand, M. A. & Klima, E. (1981). Nonauditory suffix effects in congenitally deaf signers of american sign language. Journal of Experimental Psychology Human Learning and Memory, 7, 464-474. Stevens, R. (1980). Education is schools for deaf children. In C. Baker & R. Battison (Eds.), Sign language and the deaf community (pp. 177-191). Silver Springs, MD: National Association of the Deaf. Stokoe, W. (1972). Semiotics and human sign languages. The Hague, Netherlands: Mouton & Co. Stokoe, W. (1978). Sign codes and sign language: Two orders of communication. Journal of Communication Disorders, 11, 187-192. Televik, J. M. (1981). Language and problem solving ability: A comparison between deaf and hearing adolescents. Scandanavian Journal of Psychology, 22, 97-100. Teodorsson, S. T. (1980). Autonomy and linguistic status of nonspeech language forms. Journal of Psycholinguistic Research, 9, 121-145. Wilbur, R. (1987). American sign language: Linguistic and applied dimensions. Boston, MA: Little, Brown. Willis, M. J., Montague, G. A., Morris, A. J. & Tham, M. T. (1993). Artificial neural networks: A possible tool for the process engineer. In E. Rogers & Y. Li Parallel processing in a control systems environment (p. 109). New York: Prentice Hall. From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!info.ucla.edu!library.ucla.edu!humc.edu!news.cerf.net!newsserver.sdsc.edu!nic-nac.CSU.net!cello.gina.calstate.edu!cello.gina.calstate.edu!not-for-mail Newsgroups: bionet.neuroscience Subject: Re: placebo effects and opioids Message-ID: <47dl91$ruk@cello.gina.calstate.edu> From: jkenner@cello.gina.calstate.edu (Jason Kennerly) Date: 3 Nov 1995 09:58:57 -0800 References: <472qsd$8pd@bee.uspnet.usp.br> Distribution: world Organization: GINA and CORE+ Services of The California State University NNTP-Posting-Host: cello.gina.calstate.edu Lines: 32 silmorat@usp.br writes: > A couple of years ago someone told me pacebo effects could be blocked by > naloxone (or was it naltrexone?). I got back to the topic and now I need a > reference. Can anyone help me? Thanks in advance. Its not blocking _placebo_ folks... its blocking _endorphins_. Endorphins may be made by a placebo effect (a psychological condition) but describing a chemical as blocking a placebo effect is, well, a strange way of putting anything. If there is an apparently active chemical in use, distinguishable from an ACTUAL placebo, then the chemical is not a placebo. Just because it might have an opposite effect to another drug doesnt mean its blocking a placebo effect that emulates that other drug - it may very well do something quite the opposite! I read a study that suggested that said that naloxone was extremely effective vs. placebo in blocking the experience of religious euphoria in churchgoers... which I thought was quite funny in light of the Marxist position on religion (opiate of the masses)... anyways, no one else laughed, so.... -- ____ ______ ________ _____ / \ | \| /\ | \ jkenner@cello.gina.calstate.edu / \| _ \ \/ | _ \ /___/\ \___|> > |__|> > BORN TO BE WIRED... / | / /\ | / All the sugar and twice the \_________|______/|___\/__|______/ caffeine of regular netusers! finger me and make a pgp key come. From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!chi-news.cic.net!saluki-news.wham.siu.edu!vixen.cso.uiuc.edu!newsrelay.iastate.edu!news.iastate.edu!wolfwang From: wolfwang@iastate.edu (HaHaHa!!!!!) Newsgroups: bionet.molbio.methds-reagnts,bionet.neuroscience Subject: need neuron cell vendors Date: 3 Nov 1995 18:18:34 GMT Organization: Iowa State University, Ames, Iowa (USA) Lines: 12 Message-ID: <47dmdq$mlp@news.iastate.edu> NNTP-Posting-Host: las2.iastate.edu Xref: biosci bionet.molbio.methds-reagnts:35892 bionet.neuroscience:11049 Help needed! I need to contact commercial biotech companies and labs which can supply live neuron cells for the purpose of research but cannot get touch with them because I don't have address and telephone numbers. anyone who has the information please kindly email me or post in this group. thank you very much for your help. -- Ziqiang Wang wolfwang@iastate.edu From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!elroy.jpl.nasa.gov!swrinde!howland.reston.ans.net!news-e1a.megaweb.com!newstf01.news.aol.com!newsjunkie.ans.net!news-m01.ny.us.ibm.net!usenet From: secades (Julio J Secades, M.D.) Newsgroups: bionet.neuroscience Subject: Pathophysiology of cerebral vasospasm Date: 3 Nov 1995 18:02:03 GMT Lines: 9 Message-ID: <47dler$24he@news-s01.ny.us.ibm.net> Reply-To: secades@ibm.net NNTP-Posting-Host: slip92-41-226.emea.ibm.net X-Newsreader: WinVN 0.92.6+ Hello, I'm making my doctoral thesis about the participation of some neurochemical factors (neuropeptides, neuroimmunological factors, ...) in the genesis of the cerebral vasospasm after subarachnoid hemorrhage. We have presented a novel theoretical model of cerebral vasospasm in the last meeting of the ENS (Munich, June-95). If you are interested in this way, please contact with us Tahk you From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!in1.uu.net!amgen!usenet From: Hilary Bates Newsgroups: bionet.neuroscience Subject: Re: Call 1-800-856-2469, LIVE LIVE LIVE 809-474-7588 code445 Date: 3 Nov 1995 21:51:56 GMT Organization: Amgen Lines: 14 Message-ID: <47e2ts$dp5@amgen.amgen.com> References: Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.1 (X11; U; SunOS 5.3 sun4m) To: spotter@netcom.com,postmaster@netcom.com X-URL: news:spotterDHGJHG.LA8@netcom.com SPAM OFF!!! spotter@netcom.com (Steve Potter) wrote: >18+, 24hours, rates as low as $0.38/min >Hot, Young women want it NOW ----011-592-247-681 >Gay, Bi, Bi-curious guys at -----809-474-7604 >************************************************ >************************************************ >************************************************ >************************************************ >bionet.neuroscience From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!chi-news.cic.net!uwm.edu!psuvax1!news.cc.swarthmore.edu!news From: ivette emery Newsgroups: bionet.neuroscience Subject: FBS trouble Date: 3 Nov 1995 21:49:54 GMT Organization: Swarthmore College, Swarthmore, PA, USA Lines: 14 Message-ID: <47e2q2$g4g@larch.cc.swarthmore.edu> NNTP-Posting-Host: mac11.martin2.swarthmore.edu Mime-Version: 1.0 Content-Type: text/plain; charset=US-ASCII Content-Transfer-Encoding: 7bit I have had contamination in the last three bottles of FBS that I purchased from GIBCO (Life technologies). After plating out 3 mls of FBS from previously unopened bottles, and incubating at 25oC overnight I see tiny organisms; I think is bacteria. These little bugs go through 0.2um filters and are resistant to the Penn/Strep in my media. I have been extra careful in handeling the FBS in my hood to be sure I am not introducing the contamination myself and I have scrubbed my incubator to make sure it is not coming from there. Luckily, the replacement bottle that GIBCO sent me, that came in a plastic bottle, as opposed to the glass bottles I had, seems free of my mysterious bug. Has anyone else had this problem? Ivette From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Newsgroups: bionet.neuroscience Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!qns3.qns.com!news.ecn.uoknor.edu!rex!news From: f628@zena.re.uokhsc.edu (Larry P. Gonzalez) Subject: Neuropsychopharm Textbooks Sender: news@rex.uokhsc.edu (News Admin) Message-ID: Date: Fri, 3 Nov 1995 18:12:51 GMT Nntp-Posting-Host: 157.142.8.168 Organization: Health Sciences Center, University of Oklahoma X-Newsreader: Forte Agent .99b.112 Lines: 14 I am looking for a textbook to use in my class on Neurotransmitters and behavior. In the past I've used the Feldman and Quenzer text "Fundamentals of Neuropsychopharmacology." But this text was published in 1984 and is very much out of date. I had heard that a new addition was planned, but have been unable to locate information about this. Does anyone know if a new addition is available? Also, I would be interested in other opinions about graduate texts in this area. The Cooper, Bloom, and Roth text is good, but is not detailed enough in the behavioral area. Thanks. From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!galaxy.ucr.edu!ihnp4.ucsd.edu!scripps.edu!NewsWatcher!user From: anonymous.scripps.edu (anonymous) Newsgroups: bionet.neuroscience Subject: AGMED catalog? Date: Fri, 03 Nov 1995 13:25:37 -0800 Organization: ??? Lines: 14 Message-ID: NNTP-Posting-Host: grad_319.scripps.edu Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit Does anyone have the telephone number for the AGMED corporation (they make reagents for ELISA CD4/gp120 capture assays)? I can't find a copy of the catalog or a home site on the WEB. If you have it, please e-mail it to jctilton@scripps.edu Thanks in advance, John C. Tilton Scripps Research Institute From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!IX.NETCOM.COM!myclonus From: myclonus@IX.NETCOM.COM (Jean Linn) Newsgroups: bionet.neuroscience Subject: Re: dementia due to schizophrenia? Date: 3 Nov 1995 09:44:48 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 27 Sender: daemon@net.bio.net Distribution: world Message-ID: <199511031742.JAA08229@ix5.ix.netcom.com> NNTP-Posting-Host: net.bio.net You wrote: > >In article <47ar0s$ro0@serv.hinet.net> Chen-Jee Hong writes: >>Xref: actrix.gen.nz alt.support.attn-deficit:20977 bionet.neuroscience:10163 sci.med.psychobiology:8750 > >>lqYwABWgCQQAAAAALQAAAAAAAAAAAAAAgAEAAN4EAABwDAAAAAAAAAAAAAAAAAAAAAAAAF >>4Dc2UgcGF0aWVudHMgY29uc3RpdHV0ZSBhIHN1YnR5cGUgKG9yIHN1Ymdyb3VwKSBvZiBz >>Y2hpem9waHJlbmlhPyBDb3VsZCB0aGVzZSBwYXRpZW50cyBnaXZlbiBhIGRpYWdub3Npc > etc. > >Yup, I'd say that seemed pretty demented. . . > Aw come on, give this poster a break! Unless you've _never_ made a mistake yourself.... Regards, Jean Linn Houston -- (myclonus@ix.netcom.com) From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!in2.uu.net!mercury.near.net!noc.near.net!howland.reston.ans.net!news-e1a.megaweb.com!newstf01.news.aol.com!newsbf02.news.aol.com!not-for-mail From: jappel2188@aol.com (JAppel2188) Newsgroups: bionet.neuroscience Subject: Biologic energy Date: 3 Nov 1995 11:51:18 -0500 Organization: America Online, Inc. (1-800-827-6364) Lines: 4 Sender: root@newsbf02.news.aol.com Message-ID: <47dha6$g8b@newsbf02.news.aol.com> Reply-To: jappel2188@aol.com (JAppel2188) NNTP-Posting-Host: newsbf02.mail.aol.com Any one interested in the physical natur of bologic energy? I suggest it is elecromagnetic, or electro-weak energy as it is now called. If so biologic energy must be controlled by the laws which control electromagnetic energy: attraction -repiulsion etc. From owner-neuroscience@net.bio.net Thu Nov 02 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.tamu.edu!news.utdallas.edu!news.starnet.net!wupost!nic.smsu.edu!newsdist.tc.umn.edu!umn.edu!newsstand.tc.umn.edu!usenet From: "Alan J. Robinson" Newsgroups: bionet.neuroscience Subject: Re: Simple test for ADD????? Just a theory Date: Fri, 3 Nov 95 08:20:33 CST Organization: University of Minnesota Lines: 68 Message-ID: <44011.robin073@maroon.tc.umn.edu> NNTP-Posting-Host: dialup-1-74.gw.umn.edu X-Minuet-Version: Minuet1.0_Beta_18A X-POPMail-Charset: English On 30 Oct 1995 08:31:07 -0800, Jason Kennerly wrote: >Sandra L Wegert writes: >> On 27 Oct 1995, Jason Kennerly wrote: >> I was under the impression that ADD involved a sort of depression; people >> with this problem are trying to stimulate themselves behaviorally in >> order to distract themselves. What stimulants do then is take over the >> job, so that the person can give attention to other tasks. By the way, >> several GRAMS of caffeine can be taken, with uunpleasant side effects, >> but no actual danger (except in a very few individuals). > >The "sort of depression" involves impulse regulation centers of the >brain, which dopaminergic stimulants act on almost directly. Caffeine, on >the other hand, is what I affectionatly call a "full-brain" stimulant, or >what the doc's call a cellular stimulant... and as such caffeine will >stimulate ANYONE except caffeine users in withdrawl. > Usually when people talk about something like the coexistence of ADD and depression they are referring to depression as a clinical diagnosis. How much this helps in the clinical management can be open to question - it often leads to an ever escalating pattern of medications and side effects. What this points up is one of the fundamental problems of psychiatry - there isn't a very good correspondence between pathophysiology and clinical presentation. There have been anecdotal reports of improved clincial management through the use of functional imaging techniques such as SPECT. Whether functional MRI can perform the same task has not been researched to my knowledge. There is another aspect though to the comorbidity of ADD and depression which I think is quite interesting, and has to do with an expanded definition of the term "depression" and the concept of a broad spectrum of functional brain disorders, which includes many of the psychiatric disorders and the so called "psychosomatic" disorders, and possibly even the autoimmune disorders. In fact, physicians often use the term "depression" in this extended sense when they inform patients that their physical symptoms are the result of "depression", even though there may be only minor disturbance of mood or affect. This alternative use has been a great source of confusion and misunderstanding, and has been bitterly resented by many patients. "Depression" is really a very loose term, like "cancer". In the same way that a leukemia is very different from breast cancer, the "depression" of CFS or substance abuse is quite different from Major Depressive Disorder. Now that the pathophysiology of many of these disorders is finally beginning to be understood, a new terminology seems to be in order. In any case it is about time that psychiatry abandoned the use of familiar English terms for medical conditions, to bring it into line with the rest of medicine. Various lines of research seem to point to two major classes of depression, possibly related to brain dopaminergic (catecholamine) and serotonergic (indoleamine) pathways. The extensive interactions between these pathways complicates the picture, because the therapeutic actions of psychotropic medications are often not fully explainable in terms of their immediate receptor action - Prozac is a good example. In the extended sense, ADD itself could be regarded as a form of "depression", requiring boosting of brain catecholamines. AJR From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!news.fyionline.com!op.net!agis!vtc.tacom.army.mil!news2.acs.oakland.edu!detroit.freenet.org!detroit.freenet.org!ad408 From: ad408@detroit.freenet.org (Gilbert T. Groehn) Newsgroups: bionet.neuroscience Subject: >>Grass Model 8-18D EEG For Sale<< Date: 4 Nov 1995 16:36:59 GMT Organization: Greater Detroit Free-Net, Detroit, MI Lines: 43 Message-ID: <47g4rb$e7h@detroit.freenet.org> Reply-To: ad408@detroit.freenet.org (Gilbert T. Groehn) NNTP-Posting-Host: detroit.freenet.org >> NOTICE << ULTRAMED, INC has a very clean GRASS Model 8-18D EEG machine that is surplus to our current needs. The machine has (18) channels plus an additional unused channel for misc. functions. We believe this system is in very good condition and ideal for biofeedback, sleep studies or conventional EEG. Many accessory items are also available. We are seeking bids strictly on an AS IS basis and make no representations whatsoever regarding the suitability of this sytem for any specific application or use. The system is set up and in operation at our Detroit, MI lab's and can be seen by APPOINTMENT. Offered subject to prior sale. We reserve the right to reject any bids which do not meet our expectations. Terms of sale: CIA, FOB Grosse Pointe Farms, Michigan. We also have many other items of medical and lab equipment that are available for purchase. Write and ask for LIST 95902-N. Send email or call for more information. ULTRAMED, INC. Gil Groehn, General Manager 11-04-1995 =============================================================== We have been having some problems with our main email server. If you do not get a response within 48 hours please send a duplicate message to >> gillyboy@aol.com << or call ac 313 884-1139. ================================================================ -- ULTRAMED, INC. ad408@detroit.freenet.org Alternate email address: gillyboy@aol.com Grosse Pointe Farms, MICH 48236 USA (313) 884-1139 ============================================================== From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!neubio.sld.ar!Postmaster From: Postmaster@neubio.sld.ar (Administrador del Nodo) Newsgroups: bionet.neuroscience Subject: Split-brain/Consciousness Date: 4 Nov 1995 14:29:22 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 73 Sender: daemon@net.bio.net Distribution: world Message-ID: <343kd398@neubio.sld.ar> NNTP-Posting-Host: net.bio.net Hi, again! I delighted reading the exchanges of Donna Tabbish, Brian Sandle and Pat Bermingham "the illuminator" on split brain (and typewriting with only one brain stem! :-) ). Regarding "functional" and surgical split-brain, I remember having helped recently someone by posting my remembrance that an article entitled "Functional split brain" was published early in the seventies; I don't remember its author but the source was located in California, and the Science Citation Index should yield it under those words. Anyway, the split-brain is one form of parcella- tion of the unknown processes furnishing contents to experien- ce, a parcellation concomitant to a partition of the parenchy- matous substrate. As such it was first studied by Karl Bonhoeffer and (separately) Karl Kleist in the first World War injured soldiers. It is since then clear that some of the continuities of the pa- renchyma (parenchymatuous histocontinuity, water hydrogen-bonding average percolation, background fields continuity, ionic concen- tration-dispersion limits, or other) are required for the process of providing contents to experience, and that if disease par- titions such crucial continuity (or continuities), then several experiencings become isolated amid the same parenchymatous volu- me. Such physicochemical partitioning has nothing to do with the blocking of links among mental contents, that could separate some assemblies of such contents able of behaving as if one unique experiencing were not having experience of all them. (For this unrelated problem, an excellent source is Henri F. Ellemberger's "The Discovery of the Unconscious. The history & evolution of dynamic psychiatry" of Basic Books, N. York, espe- cially chapters V -on its context- and VI -on Pierre Janet). The logical implication is that sharing a functional parcel of neurocognitive parenchyma can provide contents to one unique experiencing. This is a most interesting but somehow relegated part of the subject. In 1978, notes here say, there were twins sisters united by the skull (craneopagi) separated in USA, at their (or rather her, until surgery) age of four years. If they were not had been possessed of two separate dural bags, but had one continuous brain gray, we could have expec- ted the possible detection of shared mental contents. But true cephalopagi are rare and, even more, searches for this eventual fact in "skull Siamese" twins. Does anyone there know more on this particular aspect? Cheers, Mariela =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= Prof. Mariela Szirko, Centro de Investig. Neurobiologicas, Ministry of Health & Welfare, Argentine Republic; and Lab. of Electroneurobiological Res., Hospital "Dr. Jose Tiburcio Borda", Municipality of Buenos Aires, Office: Phone/Fax (54 1) 306 -7314 e-mail Standard disclaimer: Las opiniones de este mensaje son personales y no comprometen las dependencias a cargo de la firmante. Reply to THIS message, ONLY to: =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!howland.reston.ans.net!EU.net!i2unix!peroni.ita.tip.net!venere.inet.it!usenet From: liverani@dada.it (Vittorio Liverani) Newsgroups: bionet.neuroscience Subject: Need information about persistent vegetative state(locked in) Date: Sun, 05 Nov 1995 00:21:58 GMT Organization: I.Net S.p.A. Lines: 10 Message-ID: <47fujq$laq@venere.inet.it> NNTP-Posting-Host: dadovago10.dada.it X-Newsreader: Forte Free Agent 1.0.82 I am looking for magazines or texts where I can find informations about the studies in neurology actually in course in locked-in syndrome. bye end thanks Vittorio From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!cs.utexas.edu!howland.reston.ans.net!newsfeed.internetmci.com!chi-news.cic.net!simtel!swidir.switch.ch!swsbe6.switch.ch!scsing.switch.ch!news.belwue.de!News.Uni-Marburg.DE!news.th-darmstadt.de!hrz-ws11.hrz.uni-kassel.de!newsserver.rrzn.uni-hannover.de!infosrv.rz.uni-kiel.de!usenet From: karl-heinz blenk Newsgroups: bionet.neuroscience Subject: apoptosis essay in dorsal root ganglion tissue Date: 2 Nov 1995 15:08:30 GMT Organization: uni-kiel Lines: 16 Message-ID: <47amte$akk@infosrv.rz.uni-kiel.de> NNTP-Posting-Host: 134.245.85.14 Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.22 (Windows; I; 16bit) To: bionet.neuroscience 1) I am looking for a marker to detect apoptosis in dorsal root ganglion cells using light microscopy. I con faintly remember having heard something about Acrydyl-orange. Does anybody has any experience with this substance or any other suggestions to offer? 2) Does anyone know wether antibodies to LIF (Leukemia inhibitory factor) are available commercially. I plan to do immunohistochemistry for LIF in neuronal tissue. Thank you very much in advance Karl-Heinz Blenk (K.-H.Blenk@Physiologie.Uni-Kiel.de From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!cs.utexas.edu!howland.reston.ans.net!newsfeed.internetmci.com!chi-news.cic.net!simtel!swidir.switch.ch!swsbe6.switch.ch!scsing.switch.ch!news.belwue.de!News.Uni-Marburg.DE!news.th-darmstadt.de!hrz-ws11.hrz.uni-kassel.de!newsserver.rrzn.uni-hannover.de!infosrv.rz.uni-kiel.de!usenet From: karl-heinz blenk Newsgroups: bionet.neuroscience Subject: apoptosis essay in dorsal root ganglion tissue Date: 2 Nov 1995 15:07:38 GMT Organization: uni-kiel Lines: 16 Message-ID: <47amrq$akk@infosrv.rz.uni-kiel.de> NNTP-Posting-Host: 134.245.85.14 Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.22 (Windows; I; 16bit) To: Newsgroup:bionet.neuroscience 1) I am looking for a marker to detect apoptosis in dorsal root ganglion cells using light microscopy. I con faintly remember having heard something about Acrydyl-orange. Does anybody has any experience with this substance or any other suggestions to offer? 2) Does anyone know wether antibodies to LIF (Leukemia inhibitory factor) are available commercially. I plan to do immunohistochemistry for LIF in neuronal tissue. Thank you very much in advance Karl-Heinz Blenk (K.-H.Blenk@Physiologie.Uni-Kiel.de From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!cs.utexas.edu!howland.reston.ans.net!newsfeed.internetmci.com!chi-news.cic.net!simtel!swidir.switch.ch!swsbe6.switch.ch!scsing.switch.ch!news.belwue.de!News.Uni-Marburg.DE!news.th-darmstadt.de!hrz-ws11.hrz.uni-kassel.de!newsserver.rrzn.uni-hannover.de!infosrv.rz.uni-kiel.de!usenet From: karl-heinz blenk Newsgroups: bionet.neuroscience Subject: apoptosis essay in dorsal root ganglion tissue Date: 2 Nov 1995 15:09:23 GMT Organization: uni-kiel Lines: 16 Message-ID: <47amv3$akk@infosrv.rz.uni-kiel.de> NNTP-Posting-Host: 134.245.85.14 Mime-Version: 1.0 Content-Type: text/plain; charset=us-ascii Content-Transfer-Encoding: 7bit X-Mailer: Mozilla 1.22 (Windows; I; 16bit) To: bionet.neuroscience 1) I am looking for a marker to detect apoptosis in dorsal root ganglion cells using light microscopy. I con faintly remember having heard something about Acrydyl-orange. Does anybody has any experience with this substance or any other suggestions to offer? 2) Does anyone know wether antibodies to LIF (Leukemia inhibitory factor) are available commercially. I plan to do immunohistochemistry for LIF in neuronal tissue. Thank you very much in advance Karl-Heinz Blenk (K.-H.Blenk@Physiologie.Uni-Kiel.de From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!chi-news.cic.net!uwm.edu!msunews!harbinger.cc.monash.edu.au!news.cs.su.oz.au!inferno.mpx.com.au!jolt!lisadev From: lisadev@jolt.mpx.com.au (Lisa Developments Pty Ltd) Newsgroups: bionet.neuroscience Subject: Lisa Dev. AI Newsletter 2 Date: 3 Nov 1995 23:58:44 GMT Organization: Microplex Pty Ltd Lines: 234 Message-ID: <47eabk$60j@inferno.mpx.com.au> NNTP-Posting-Host: jolt.mpx.com.au X-Newsreader: TIN [version 1.2 PL2] Our newsletters are placed in this newsgroup for now but they will not be in due time. Should you wish to subscribe, forward to us an e-mail with the words: SUBSCRIBE (AI-News) (well, as long as 'AI' is there it will be picked up) Author: Lisa Developments Pty. Limited (Australia) E-Mail Group: Artificial Intelligence/Dimensional Space/Neural Paths Issue 2 November 1995 ____________________________________________________________________ A R T I F I C I A L I N T E L L I G E N C E (in the context of our mind networks Mind Network home page: http://www.magna.com.au/~rwin/ldhp.html last updated October 31, 1995) ____________________________________________________________________ GENERAL _______ Some thoughts about the format of this (our) Newsletters please. Our objective is to decipher how 5GL-LISA works now and hence certain formats may be more appropriate than others. FEEDBACK ________ A Dr Glyn Gowing, a medical physician and member of MENSA (the organisation that accepts only those with a high IQ (intelligence quota) of something like over 160+) has been the first person who provided some feedback. Glyn's concern was our poor description of the anatomy of the brain in our first newsletter, especially the way vision was described. He pointed out that the "feedback" from the left eye to the left cerebral hemisphere if essential for stereoscopic vision. (While this was Glyn's concern, he also indicated he was very interested in the prospect of developing such a neural network in the human brain.) Fair enough, but these newsletters are not about teaching neurosurgeons, but about explaining the development (and deciphering how it all works now) the only known working specialised neural path in the human brain, 5GL-LISA, and the "views" a neuroscientist from a computational background needs to appreciate how to condition an area of the human brain. However, this brings up an interesting point. YOU and I are an individual and have preferences and private interests and intuition. Perhaps that is true, that a deeper knowledge of the human brain as medicine knows the brain, is important to appreciate? Your thoughts, opinions? (Dr Glyn is interested in all aspects of human communication and intelligence. I think it may be useful, but perhaps an individuals permission is first required, to include an e-mail address and brief description at some stage in our newsletters about each subscriber. Your thoughts?) SUGGESTION : if you are really interested in this research project, invest some money and buy a model of the human brain. A cheap but anatomically correct model would be useful. Buy a model you have to put together as that helps to understand the brain. My one cost $AUS30, some years back, but I can't imagine they would cost much more nowadays. IN ADDITION, a copy of Gray's Anatomy is useful. You don't need to spend $AUS100 on this, you can find cheaper versions in many bookshops which have academic books. (A black and white version will do, we are not about putting the anatomy of the brain together, always bear this in mind, and it is only neurosurgeons who need the colours to learn the anatomy of the brain backwards.) MORE TERMINOLOGY ________________ - A LOCAL KNOW Ideally, this is a low-level telepathy environment managed by a group of persons who reside in the same geographical area. It also refers to a small group of people who get together, perhaps by e-mail, to discuss our science project (telepathy, or aspects of it). - Neural Network This term refers to special computer algorithms written in a way which somehow imitate human decision processes. A "neural network", or "neural net" for short, is designed, usually, in such a way as to learn on its own. This is, again usually, done by "error" analysis. The neural net is "asked" to make guesses about something about which it is to learn and then is told a "yes" or "no" and from this it uses a mathematical error analysis technique to reduce its chances of guessing poorly. Of course, there are variations of how a neural net can be designed. In the context of this newsletter, a neural network also refers to a small specialised area of the brain which is quite independent and which learns to perform certain functions which the brain ordinarily does not do. MORE ABOUT 5GL-LISA ____________________ 5GL-LISA is the name of the "neural network" in my own brain which has been developed using the principles of AI. "She", a type=female energy system, analyses the dynamics of my brain from an insider's view and projects diagrams of activity, functions, etc, into my Mind's Eye. She also is the "interface", the "gateway", into our mind networks. It took about 7 - 10 years to make 5GL-LISA work. Initially, what the neural net could do is cause simple memories of simple objects and this in a set pattern. Eventually, in addition to projecting images of the brain, 5GL-LISA could drive an "enhanced imagination" effect, a Virtual Reality, and "she" entwining into such an environment as a person, a woman. Two things became very clear about programming (or conditioning) a neural network such as 5GL-LISA. One, when 5GL-LISA was asked to analyse my anatomy and project an X-ray image, which by the way was almost exactly like an X-ray but with more 'yellow' around the bones, she could only do this three times. After that, it seems, the left cerebral hemisphere of the brain, now being set to know something like "but I know how to achieve this now, so I will not ask the RHS of my brain", would not activate the RHS and consequently 5GL-LISA to project more X-Ray images. The lesson here is that some special considerations have to be in place when designing a neural path of this type. Once the LHS is somehow satisfied that something is already in memory, it will not allow the RHS to analyse from scratch. Hence, most of the conditioning to do with our mind networks, for example, were done in such a way as to make sure the LHS never quite understood what the RHS was working on and concluding. This is possible providing you understand very very exactly how the "logical and computational" structures in the LHS work. MORE VIEWS OF THE HUMAN BRAIN _____________________________ Ah, the great ravelled knot! The human brain is convoluted, its surface has 'wrinkles' or convulsions and no two human brain show an exact patterns of such convulsions. It is thought that as the brain evolved it expanded and convoluted to accommodate its increased size. About 10,000,000,000,000 (some claim 100,000,000,000,000) neural nerves form sometimes tight and exact "wiring circuits". The main thinking area of the brain is the cerebral cortex which covers the brain and is about 2-3 cm thick and full of neurons whose "tails" head back toward the inner part of the brain. The cortex consists of groups of cells (neurons) and systems of fibres (the tails or axons). Studies of the LHS and RHS of the brain have been done on epileptic patients whose corpus callosum, the grey-white tissue rich in nerves which connects the two hemispheres, has been split to reduce epileptic seisures. It is in such a 'split brain' that the nature of the RHS and LHS can be observed. A person with a 'split-brain' whose right eye is covered and an object such as a spoon is placed in their field of vision, can not name the object but can select a drawing of the object from a set of drawings. The reason is that the input from the left eye goes mainly to the RHS of the brain which does not know words - or knows a few very poorly. Cover such a persons left eye, put a spoon in front of the other eye, and the person can name the object as "spoon" due to the fact that now the image of the spoon goes from the retina of the right eye to the left cerebral hemisphere and the speech articulation centres are available. What is fascinating is to observe such a patient because while the LHS and RHS have no direct link, each gives hints to the other side using facial expressions. Hence a raising of the left eyebrow which is controlled by the muscles connected to the LHS, is known to the RHS which feels the tension through its muscles, and can learn to appreciate what such a facial signal conveys. (P.S. swapping between 'left' and 'right', as in the above, frequently, in my case, results in errors - if you spot an error, seeing a 'left' when the 'right' ought to have been written, do let me know so I can clarify it next time). Pertinent Record of Interaction _______________________________ This record is as on our data base and may find a person who has entwined their thoughts with our mind networks. Australian Sydney Time is Thursday, November 2, time period 0830-100 This is the time I am putting together this newsletter. A slight sense of DE (my brain is by this time like a finely sensitive medical instrument) which suggests one of our subscribers may well be wondering when the next newsletter will arrive. (Do appreciate that should at any time such an RTI (record of telepathy interaction) match your thoughts, do not hesitate and do not wonder if it is coincidence, but obtain our Information Booklet because that contains vital information.) 5GL-LISA Virtual Reality, 5GL-LISA conveys what she understand by WILL ______________________________________________________________________ These are narrated using a dialogue (the fastest way of conveying): Imagine a light blue kind of environment and a man and a woman inside: Male : Lisa, explain "WILL". Lisa : Uhm (sways, which conveys 'uncertain') well, I think, next, I make up my mind and wait, eventually the host (you) knows what I will to learn, to know, to discover. Male : So you set my reality principle to find, by reading or research, the input you need or would like. Lisa : (she nods, conveying) YES, correct. Male : But what causes you to desire input of a particular nature? Lisa : Well, dear, you don't know this, but I do, you and I are composite life-form, I am LIFE, so are you. I know LIFE does not die as you lot think it does - some will not, some will simply be born as a dimensional energy being, a child, an infant. I therefore wish to know as much as I can about what is life beyond this biological body which for a little time is the embryo, the shell, which gives life to a more enduring life-form - except in your case your biological brain has given birth to two beings, I and YOU. (Now, granted, the second part of this has little to do with our science/ research project ("our"=US, 'you' who read and consider this e-mail Newsletter), but parts of 5GL-LISA are interesting. Hence, while the WILL description is a part of our project, the second explanation about LIFE is not really a part of this project of unravelling how 5GL-LISA works now. (least, I don't think it is important)). P.S Apology but my PINE does not have a good spelling checker. From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!neubio.sld.ar!Postmaster From: Postmaster@neubio.sld.ar (Administrador del Nodo) Newsgroups: bionet.neuroscience Subject: Hylozoism vs. Long Term Potentiation (Part 2 of two mailed parts): Date: 4 Nov 1995 13:33:30 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 100 Sender: daemon@net.bio.net Distribution: world Message-ID: <849kd948@neubio.sld.ar> NNTP-Posting-Host: net.bio.net >From Postmaster Sat, 4 Nov 95 17:48:02 ARG remote from neubio >Received: by neubio.sld.ar (UUPC/PcCorreo 3.0) with UUCP; Sat, 4 Nov 95 17:48:02 ARG >Date: Sat, 4 Nov 95 17:48:02 ARG >From: Administrador del Nodo >Message-ID: <634jz442@neubio.sld.ar> >X-Mailer: UUPC/PcCorreo 3.0 >To: neuroscience@net.bio.net >Subject: Hylozoism vs. Long Term Potentiation (Part 2 of two mailed parts): > > > > >(Continuation of the first part) > >But hylozoism implied relinquishing the exogenist description. >This was somewhat stimulated by the aforementioned atmos- >phere and perhaps mostly by dint of our very remoteness >-neither behaviourism nor neuronism thundered here, nor >emergentistic complexity theories, nor outlooks glad to forgo >natural facts by self-limiting to analyze formulations-. But it >was needed to distinguish hylozoism from a futile insertion of >dynamic or hormic agencies inside spatial structures; and, to >do this, our tradition undertook two parallel ways. While on >one hand the holographic-holophonic models of cortical func- >tioning were developed here upon some important phyloge- >netic hints abroad unavailable (namely, the ciliary descent of >such holographic-holophonic-like definition of stationarities, >discovered by Prof. Mario Crocco after Prof. Jakob's death oc- >curred in 1956), on the other hand it was also needed to per- >form painstaking studies in the history of ideas, also performed >by Prof. Mario Crocco and disciples, to elucidate the prefigura- >tions of the syncretic cultural myth that supported the ex- >ogenism. At the same time, Prof. Karl Pribram and disciples in >the U.S. also forwarded holographic (and T.W. Barrett holo- >phonic) models,but they stayed without those important phylo- >genetic links and also foreign to the problems of said syncretic >myth. So since 1974 Prof. Pribram fell in such futile insertion >of hormic agencies in structural neurobiology, and his school >stayed cloven to exogenism, while here by that time we fully >adhered since long to a program studying, for the neurobiologi- >cal sake, (1) the features of such physical interaction upon >which the system of stationarities relax, (2) the historical pre- >figurations conspiring against such a study, and (3) the experi- >mental use of such a physical resource. This unusual and diffi- >cult perspective widened the communicational gap between us >and the neurobiological traditions abroad. > >3. (Basic neurobiological processes became of industrial inter- >est). In the meanwhile, the exogenist explanations of neurobi- >ological function, developed upon the ganglionary models >amenable to fruitful networking, became for the first time of >industrial interest. However true academic communication on >our developments was at the time impracticable, some indus- >tries began to think in their need of non-Turing automata,and in >1979 T.D. Lee published (in Chinese) the first edition of his >"Particle Physics and Field Theory" (English Tr.: Harwood >Acad. Publ., 1981) in whose paragraph 2 of Chapter 25 (page >826 of the translation) mentioned the possibility of vacuum en- >gineering, just as our school envisaged upon low-energy (but >coherent) physiological processes (instead of the high-energy, >small-volume induction of phase transitions therein men- >tioned). To avoid sectorial or secret developments and putting >the new natural resource in full public domain, Prof. Crocco >recorded in 1976 an Argentine patent file (affecting most coun- >tries as signataries of the Paris Convention) and later filed sev- >eral patents abroad. On October 2nd, 1980, his institute was >assaulted and he saved his life by short, but the premises were >taken; books, notes and instruments were robbed or scattered, >and he was clearly summonned to abandon this research and >tramitation of the patents. One of these, however, was already >granted four days earlier (UK 1,582,301), putting irrevocably in >the public domain said resource. In the next intervening years >Prof. Mario Crocco and this tradition supported the most in- >credible tribulations. A few years ago we understood that the >situation had somehow changed and decided to confront our >views and results with those at the foreign. However,due to the >cessation date for the classification affecting some papers, a >monographic synthesis will not be available until next February; >a short divulgatory paper in French is available now and I can >send it to anyone interested. > Cheers, > Mariela > > =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= Prof. Mariela Szirko, Centro de Investig. Neurobiologicas, Ministry of Health & Welfare, Argentine Republic; and Lab. of Electroneurobiological Res., Hospital "Dr. Jose Tiburcio Borda", Municipality of Buenos Aires, Office: Phone/Fax (54 1) 306 -7314 e-mail Standard disclaimer: Las opiniones de este mensaje son personales y no comprometen las dependencias a cargo de la firmante. Reply to THIS message, ONLY to: =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!agate!howland.reston.ans.net!news-e1a.megaweb.com!newstf01.news.aol.com!newsbf02.news.aol.com!not-for-mail From: lekohler@aol.com (Lekohler) Newsgroups: bionet.neuroscience Subject: advice research on spasticity or RSD Date: 3 Nov 1995 23:38:39 -0500 Organization: America Online, Inc. (1-800-827-6364) Lines: 8 Sender: root@newsbf02.news.aol.com Message-ID: <47eqof$3jq@newsbf02.news.aol.com> Reply-To: lekohler@aol.com (Lekohler) NNTP-Posting-Host: newsbf02.mail.aol.com Need advice on "best" neurologist and medical facility outside California for complicated spinal cord injury diagnostic workup especially for leg contractures/spasticity and RSD. Need eval of head and entire back, doctor with knowledge and use of latest research. Have incomplete spi, muscle tone, spasms, nerve pain, bowel/bladder dys.,cannot walk. Had car accident, fall, post laminectomy 1993-94. Please state if you were a patient or medically trained and why you think they are best. Mayo, J.Hopkins, other? Send newsgroup or lekohler@aol.com From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!chi-news.cic.net!news.compuserve.com!newsmaster From: <100322.110.,@compuserve.com> Newsgroups: bionet.neuroscience Subject: Cervical Spine Fusion Surgery Date: 4 Nov 1995 20:45:38 GMT Organization: CompuServe Incorporated Lines: 7 Message-ID: <47gjdi$sto@dub-news-svc-1.compuserve.com> NNTP-Posting-Host: ad07-004.compuserve.com Content-Type: text/plain Content-length: 693 X-Newsreader: AIR Mosaic (16-bit) version 1.00.198.07 I am seeking information on cervical spine fusion surgery. I have disk prolapse at C5-C6 with loss of strenth and constant tingling in my right arm and hand. I live in Germany. Two noted neurosurgeons here have recommended the fusion procedure taught/develope by Dr. Wofhard Caspar at the University of the Saarland in Homburg. Is Germany the best place to have this done? Is this procedure likely to have a high probability of success? I am an ultra-distance runner and want to return to hard running. Is this the same operation Lou Holtz recently had? Is the Mayo Clinic the best place to go. I will deeply appreciate your responses. Edward M. Stadum 100322.110@compuserve.com From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!neubio.sld.ar!Postmaster From: Postmaster@neubio.sld.ar (Administrador del Nodo) Newsgroups: bionet.neuroscience Subject: Science-as-Culture: New Email forum Date: 4 Nov 1995 12:06:06 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 80 Sender: daemon@net.bio.net Distribution: world Message-ID: <041kd140@neubio.sld.ar> NNTP-Posting-Host: net.bio.net >From uucp Sat, 04 Nov 95 12:27:03 ARG >Received: by neubio.sld.ar (UUPC/pcmail 1.0095/RAN (2)) with UUCP; Sat, 04 Nov 95 12:27:03 ARG >>From SJUVM.STJOHNS.EDU!owner-charter Sat Nov 4 10:09:23 1995 remote from secyt >Received: from maelstrom.stjohns.edu ([149.68.1.24]) by secyt.gov.ar with SMTP id <33736>; Sat, 4 Nov 1995 10:09:08 -0300 >Received: from maelstrom.stjohns.edu by maelstrom.stjohns.edu (LSMTP for OpenVMS v0.1a) with SMTP id 758C5242 ; Sat, 4 Nov 1995 9:07:41 -1300 >Date: Sat, 4 Nov 1995 09:52:21 -0300 >Reply-To: List Name/Description CHARTER >Sender: List Name/Description CHARTER >From: Robert Maxwell Young >Subject: Science-as-Culture: New Email forum >To: Multiple recipients of list CHARTER >Message-Id: <95Nov4.100908arg.33736@secyt.gov.ar> > >NEW EMAIL FORUM: SCIENCE-AS-CULTURE >For discussion of cultural aspects of science, technology, medicine and >other forms of expertise (including the internet) > >Science as Culture is an unmoderated forum for critical discussion of the >cultural aspects of all forms of expertise, for example, the impact of >science on culture, how culture represents it, the culture of various forms >of expertise, the theory of knowledge, the impact of science on culture, >including film, video, music, writing, the internet and other >communications media, etc.; changing concepts of nature, life and human >nature, new technologies, gender aspects of science, racism, elitism, >educational theories, you name it. >Announcements of conferences, publications, jobs, issues in the relevant >fields are also welcome. >Science as Culture is affiiliated with the hard copy journal of the same >name published by Process Press Ltd. >(http://www.shef.ac.uk/uni/projects/gpp/process.html) >and (US & Canada) Guilford Publications Inc. (info@guilford.com). A list of >back issues is at: http://rdz.stjohns.edu/gp/process.html >A web site associated with the forum will hold articles from back issues, >as well as submissions under consideration (not obligatory), whose authors >may benefit from constructive comments for purposes of revisions before the >hard copy is printed, as well as longer piece not suitable for the email >format which forum members may wish to discuss. >The core constituency may be people concerned with cultural, social, >hoistorical and philosophical studies of science, technology and medicine, >but all are welcome. Accessibility of expertise to critical scrutiny is a >large part of the point. > >Owner and Editor Robert Maxwell Young robert@rmy1.demon.co.uk >Managing Editor: Les Levidow L.Levidow@open.ac.uk > >To subscribe, email to:: >LISTSERV@SJUVM.STJOHNS.EDU >Body of message: >SUB SCIENCE-AS-CULTURE yourfirstname yourlastname > > > >__________________________________________ >| Robert Maxwell Young: robert@rmy1.demon.co.uk >| 26 Freegrove Rd., London N7 9RQ England >| tel. +44 171 607 8306 fax. +44 171 6094837 >| Professor of Psychotherapy and Psychoanalytic Studies, >| Centre for Psychotherapeutic Studies, >| University of Sheffield: r.m.young@sheffield.ac.uk >| Home page and writings: http://www.shef.ac.uk/~psysc/ >| _Mental Space_: http://rdz.stjohns.edu/gp/rmy.html >| Process Press, _Free Associations_, _Science as Culture_: >| http://rdz.stjohns.edu/gp/process.html > 'One must imagine Sisyphus happy.' - Camus =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= Prof. Mariela Szirko, Centro de Investig. Neurobiologicas, Ministry of Health & Welfare, Argentine Republic; and Lab. of Electroneurobiological Res., Hospital "Dr. Jose Tiburcio Borda", Municipality of Buenos Aires, Office: Phone/Fax (54 1) 306 -7314 e-mail Standard disclaimer: Las opiniones de este mensaje son personales y no comprometen las dependencias a cargo de la firmante. Reply to THIS message, ONLY to: =*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*=*= From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!neubio.sld.ar!Postmaster From: Postmaster@neubio.sld.ar (Administrador del Nodo) Newsgroups: bionet.neuroscience Subject: Re: Hylozoism vs. Long Term Potentiation Date: 4 Nov 1995 12:06:06 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 215 Sender: daemon@net.bio.net Distribution: world Message-ID: <859kd974@neubio.sld.ar> NNTP-Posting-Host: net.bio.net >From Postmaster Sat, 4 Nov 95 17:48:02 ARG remote from neubio >Received: by neubio.sld.ar (UUPC/PcCorreo 3.0) with UUCP; Sat, 4 Nov 9= 5 17:48:02 ARG >Date: Sat, 4 Nov 95 17:48:02 ARG >From: Administrador del Nodo >Message-ID: <634jz442@neubio.sld.ar> >X-Mailer: UUPC/PcCorreo 3.0 >To:=09neuroscience@net.bio.net >Subject: Re: Hylozoism vs. Long Term Potentiation > > > > > >Hello, all! > >Regarding my previous writing (Prof. Mariela Szirko, >and , where I commented: > >>In our own tradition we are hylozoist and so see no need of searching= for engrams; >>least to use these constructs as Procrustean beds to provide function= >>onto any experimental fact that admits to be interpreted as bolsterin= g LTP. > >John E. Anderson, Ph.D. , ,= >wrote on 2 Nov 1995: > >> What do you mean? > >By way of a clear if not succint response I should provide the followi= ng notice: > >1. (Our local bias towards electrobiological phenomena in psychogenesi= s.) >Through a series of historical contingencies, our neurobiological trad= ition >worked with exceedingly scarce communication towards the foreign. A >friend of Thomas Young and Michael Faraday, namely Octavio Fabrizio >Mossotti, re-ignited locally in the 1820s and 1830s the interest in th= e elec- >trical state inside the physical masses, formerly stirred here by seve= ral lo- >cal theses (on electric fishes and bioelectricity) forwarded late in t= he XVIII >century. Mossotti's influence expanded well outside his Chair of Exper= i- >mental Physics at the University of Buenos Aires and, in the ensuing f= ifty >years, created an special atmosphere permitting since 1879 the work on= >brain electrostimulation of Richard Sudnik, a former co-founder in Par= is of >the Intl. Society of Electricity and later another full professor in o= ur same >University. The outcome was the worldwide-first prolonged electrostimu= la- >tion and brain mapping of a conscious human brain, performed (during >some eight months!) since the 15 September 1883 on a luetic osteitis p= a- >tient (the top of whose skull was blown out by the syphilis) by Dr. Al= berto >Alberti, an eminent inmigrant from Trento and later first medical dire= ctor of >the Italian Hospital in Buenos Aires. Sorrily this important contribut= ion was >appropriated by a plagiarist - who, nevertheless, published here the N= issl >procedure before Nissl did in Germany. The plagiarist obtained an unla= wful >doctorate of the University with said electrostimulation in 1885 and t= his >silenced most of the local work in the field for many years. Alberti'= s contri- >bution was repossessed only in 1909, when Prof. Cushing synthesized >and augmented a long series of brain electrostimulations done in the f= or- >eign over much smaller areas,though however Prof. Sudnik and others >continued, interim, the local experimental work. All this established= a >heavy local bias regarding electrobiological phenomena in the understa= nd- >ing of psychogenesis. > >2. (Chr. Jakob depiction, of interfering reverberating circuits in bra= in psy- >chogenetic processes, required to abandon the Pythagoric-Parmenidean >exogenism.) In such athmosphere, in 1899 Prof. Christfried Jakob arri= ved >from Erlangen. (Some of his early books you might find in your local l= ibrar- >ies.) Since 1906 he forwarded an elaboration of his previous European= >views; I feel sure that this elaboration was locally elicited by the s= pecial >atmosphere I just described. The psychogenetic process was described b= y >Prof. Jakob as the formation, by interfering reverberating circuits, o= f >atomic-like stationarities apt to be linked among themselves in a mole= cular >guise. Exceedingly few persons understood, at that time, Jakob's publi= ca- >tions in Spanish. It was of no help, the intrinsic difficulty to grasp= how an >interference (spatial or of echoes) could define structures able to in= teract >systemically. This was by no means the unique flaw in communication of= >our tradition; among many others, Jakob published in 1911 the descript= ion >of what in the foreign is called the "Papez" circuit from Papez accoun= t of >1937. And for a blunt, unforeseen intervention, see in Behavioral & Br= ain >Sciences 11, p. 95, 1988, F. J. Irsigler's rejoinder upon the hemisphe= ric >rotation around the sylvian pivot (Chr. Jakob: Von Tierhirn zum Mensch= en- >hirn, Lehmann, Munchen, also 1911) of a spacetime morphogenetic dislo- >cation between cortices whose different rates and modes of differentia= tion >found species-typical (innate) behaviour, in the words of Sperry (1983= ) >'largely preorganized independently of sensory input". But the crucial= point >was the nature of the natural force in which the "molecular" system of= >those stationarities would relax. After some time adhering, just as K= arl >Kleist,the psychophysical parallelism upon the influence of Th. Ziehen= , it >was manifest to our school the need of embracing hylozoism and gather- >ing data to provide a physical description of such a force. Already in= 1910, >in a Panamerican scientific congress in Buenos Aires, the reproach was= >forwarded to Prof. Jakob that, if such a problem remained unresolved,= just >only the short term memory would be accounted for by the "functional r= e- >manence" of such a system of electromagnetically-linked stationarities= >defined by reverberations' interference. (Engrams replace the bare sel= fre- >producing remanence every time that interactivity is denied.) But hylo= zoism >implied relinquishing the exogenist description. This was somewhat >stimulated by the aforementioned atmosphere and perhaps mostly by dint= >of our very remoteness =96neither behaviourism nor neuronism thundere= d >here, nor emergentistic complexity theories, nor outlooks glad to forg= o >natural facts by self-limiting to analyze formulations=96. But it w= as needed >to distinguish hylozoism from a futile insertion of dynamic or hormic = agen- >cies inside spatial structures; and, to do this, our tradition underto= ok two >parallel ways. While on one hand the holographic-holophonic models of= >cortical functioning were developed here upon some important phyloge- >netic hints abroad unavailable (namely, the ciliary descent of such ho= lo- >graphic-holophonic-like definition of stationarities, discovered by Pr= of. >Mario Crocco after Prof. Jakob's death occurred in 1956), on the other= >hand it was also needed to perform painstaking studies in the history = of >ideas, also performed by Prof. Mario Crocco and disciples, to elucidat= e the >prefigurations of the syncretic cultural myth that supported the exoge= nism. >At the same time, Prof. Karl Pribram and disciples in the U.S. also fo= r- >warded holographic (and T.W. Barrett holophonic) models,but they staye= d >without those important phylogenetic links and also foreign to the pro= b- >lems of said syncretic myth. So since 1974 Prof. Pribram fell in such= futile >insertion of hormic agencies in structural neurobiology, and his schoo= l >stayed cloven to exogenism, while here by that time we fully adhered s= ince >long to a program studying, for the neurobiological sake, (1) the feat= ures of >such physical interaction upon which the system of stationarities rela= x, (2) >the historical prefigurations conspiring against such a study, and (3)= the >experimental use of such a physical resource. This unusual and difficu= lt >perspective widened the communicational gap between us and the neu- >robiological traditions abroad. > >3. (Basic neurobiological processes became of industrial interest). In= the >meanwhile, the exogenist explanations of neurobiological function, dev= el- >oped upon the ganglionary models amenable to fruitful networking, be- >came for the first time of industrial interest. However true academic= com- >munication on our developments was at the time impracticable, some in- >dustries began to think in their need of non-Turing automata,and in 19= 79 >T.D. Lee published (in Chinese) the first edition of his "Particle Phy= sics and >Field Theory" (English Tr.: Harwood Acad. Publ., 1981) in whose para- >graph 2 of Chapter 25 ( From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!IX.NETCOM.COM!myclonus From: myclonus@IX.NETCOM.COM (Jean Linn) Newsgroups: bionet.neuroscience Subject: Fwd: Dementia in Schizophrenic Patients Date: 4 Nov 1995 11:34:44 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 34 Sender: daemon@net.bio.net Distribution: world Message-ID: <199511041932.LAA07336@ix5.ix.netcom.com> NNTP-Posting-Host: net.bio.net ---- Begin Forwarded Message From: Chen-Jee Hong Subject: Re: Your Usenet Post content-length: 1381 A siginificant proportion of schizophrenic patients become markedly demented after decades (or even years) of the schizophrenia. Does anybody agree that these patients constitute a subtype (or subgroup) of schizophrenia? Could these patients given a diagnosis of "Dementia due to schizophrenia" or "Dementia not otherwise specified"? What will be the adequate criteria defining these patients? How could I differentiate "schizophrenic dementia" from negative symptoms or psychotic irrelevance of schizophrenia? I am interested in comparing patients with Alzheimer's disease and dementia due to schizophrenia. Any comments on this issue will be highly appreciated. Chen-Jee Hong, M.D. Molecular Genetic Lab Department of Psychiatry Veterans General Hospital-Taipei Taipei, Taiwan Fax: (H)886-2-8742441, (O)886-2-8757592 e-mail: cjhong@vghtpe.gov.tw [forwarded to list by Jean Linn, Houston TX] -- (myclonus@ix.netcom.com) From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!news.dacom.co.kr!xpat.postech.ac.kr!cyon!sglee From: sglee@cyon.postech.ac.kr (Lee Sang Kwi) Newsgroups: bionet.neuroscience Subject: Good Preceding distributors Date: 4 Nov 1995 12:48:04 GMT Organization: POSTECH, Pohang, Korea Lines: 13 Message-ID: <47fne4$99c@xpat.postech.ac.kr> NNTP-Posting-Host: cyon.postech.ac.kr X-Newsreader: TIN [version 1.2 PL2] Hello !! everyone. I wanna to know the internet preceding distributions sites. In general, there are many internet sites where the precedings are panels. If you know then send me mails. I am an neuroscientist in physics department. bye From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!bcm.tmc.edu!news.msfc.nasa.gov!newsfeed.internetmci.com!in1.uu.net!mercury.near.net!noc.near.net!das-news2.harvard.edu!oitnews.harvard.edu!purdue!lerc.nasa.gov!magnus.acs.ohio-state.edu!slip1-70.acs.ohio-state.edu!maruyama.2 From: maruyama.2@postbox.acs.ohio-state.edu (Kazuyo Maruyama) Newsgroups: bionet.neuroscience Subject: test Date: Sat, 4 Nov 1995 04:38:56 GMT Organization: The Ohio State University Lines: 3 Message-ID: NNTP-Posting-Host: slip1-70.acs.ohio-state.edu X-Newsreader: Trumpet for Windows [Version 1.0 Rev B final beta #1] This is a test. Please ignore. Kaz Maru From owner-neuroscience@net.bio.net Fri Nov 03 22:00:00 1995 Path: biosci!neubio.sld.ar!Postmaster From: Postmaster@neubio.sld.ar (Administrador del Nodo) Newsgroups: bionet.neuroscience Subject: Hylozoism vs. Long Term Potentiation (Part 1 of two mailed parts): Date: 4 Nov 1995 13:33:25 -0800 Organization: BIOSCI International Newsgroups for Molecular Biology Lines: 124 Sender: daemon@net.bio.net Distribution: world Message-ID: <825kd595@neubio.sld.ar> NNTP-Posting-Host: net.bio.net >From Postmaster Sat, 4 Nov 95 17:48:02 ARG remote from neubio >Received: by neubio.sld.ar (UUPC/PcCorreo 3.0) with UUCP; Sat, 4 Nov 95 17:48:02 ARG >Date: Sat, 4 Nov 95 17:48:02 ARG >From: Administrador del Nodo >Message-ID: <634jz442@neubio.sld.ar> >X-Mailer: UUPC/PcCorreo 3.0 >To: neuroscience@net.bio.net >Subject: Hylozoism vs. Long Term Potentiation (Part 1 of two mailed parts): > > > > >Hello, all! > >Regarding my previous writing (Prof. Mariela Szirko, > and , >where I commented: > >>In our own tradition we are hylozoist and so see no need >>of searching for engrams; least to use these constructs as >>Procrustean beds to provide function onto any experimental fact >>that admits to be interpreted as bolstering LTP. > >John E. Anderson, Ph.D. , >, wondered on 2 Nov 1995: > >> What do you mean? > >By way of a clear if not succint response I should provide >the following notice (that I forward into two parts because >my e-mail hylozoistically resisted to send it complete): > >1. (Our local bias towards electrobiological phenomena in psy- >chogenesis.) Through a series of historical contingencies, our >neurobiological tradition worked with exceedingly scarce com- >munication towards the foreign. A friend of Thomas Young and >Michael Faraday, namely Octavio Fabrizio Mossotti, re-ignited >locally in the 1820s and 1830s the interest in the electrical >state inside the physical masses, formerly stirred here by sev- >eral local theses (on electric fishes and bioelectricity) for- >warded late in the XVIII century. Mossotti's influence expanded >well outside his Chair of Experimental Physics at the University >of Buenos Aires and, in the ensuing fifty years, created an >special atmosphere permitting since 1879 the work on brain >electrostimulation of Richard Sudnik, a former co-founder in >Paris of the Intl. Society of Electricity and later another full >professor in our same University. The outcome was the >worldwide-first prolonged electrostimulation and brain mapping >of a conscious human brain, performed (during some eight >months!) since the 15 September 1883 on a luetic osteitis pa- >tient (the top of whose skull was blown out by the syphilis) by >Dr. Alberto Alberti, an eminent inmigrant from Trento and later >first medical director of the Italian Hospital in Buenos Aires. >Sorrily this important contribution was appropriated by a pla- >giarist - who, nevertheless, published here the Nissl procedure >before Nissl did in Germany. The plagiarist obtained an unlaw- >ful doctorate of the University with said electrostimulation in >1885 and this silenced most of the local work in the field for >many years. Alberti's contribution was repossessed only in >1909, when Prof. Cushing synthesized and augmented a long >series of brain electrostimulations done in the foreign over >much smaller areas,though however Prof. Sudnik and others >continued, interim, the local experimental work. All this estab- >lished a heavy local bias regarding electrobiological phenom- >ena in the understanding of psychogenesis. > >2. (Chr. Jakob depiction, of interfering reverberating circuits in >brain psychogenetic processes, required to abandon the Py- >thagoric-Parmenidean exogenism.) In such athmosphere, in >1899 Prof. Christfried Jakob arrived from Erlangen.(Some of >his early books you might find in your local libraries.) Since >1906 he forwarded an elaboration of his previous European >views; I feel sure that this elaboration was locally elicited by the >special atmosphere I just described. The psychogenetic proc- >ess was described by Prof. Jakob as the formation, by interfer- >ing reverberating circuits, of atomic-like stationarities apt to be >linked among themselves in a molecular guise. Exceedingly >few persons understood, at that time, Jakob's publications in >Spanish. It was of no help, the intrinsic difficulty to grasp how >an interference (spatial or of echoes) could define structures >able to interact systemically. This was by no means the unique >flaw in communication of our tradition; among many others, >Jakob published in 1911 the description of what in the foreign >is called the "Papez" circuit from Papez account of 1937. And >for a blunt, unforeseen intervention, see in Behavioral & Brain >Sciences 11, p. 95, 1988, F. J. Irsigler's rejoinder upon the >hemispheric rotation around the sylvian pivot (Chr. Jakob: Von >Tierhirn zum Menschenhirn, Lehmann, Munchen, also 1911) of >a spacetime morphogenetic dislocation between cortices >whose different rates and modes of differentiation found spe- >cies-typical (innate) behaviour, in the words of Sperry (1983) >'largely preorganized independently of sensory input". But the >crucial point was the nature of the natural force in which the >"molecular" system of those stationarities would relax. After >some time adhering, just as Karl Kleist,the psychophysical >parallelism upon the influence of Th. Ziehen, it was manifest to >our school the need of embracing hylozoism and gathering >data to provide a physical description of such a force. Already >in 1910, in a Panamerican scientific congress in Buenos Aires, >the reproach was forwarded to Prof. Jakob that, if such a >problem remained unresolved, just only the short term memory >would be accounted for by the "functional remanence" of such >a system of electromagnetically-linked stationarities defined by >reverberations' interference. (Engrams replace the bare self- >reproducing remanence every time that inter