THE FAUCI FILES, Vol 3( 26): HIV Cocktail Warnings Ignored From H.A.A.R.T. to F.A.A.R.T.
fredshaw at primenet.com
Tue Feb 1 02:40:27 EST 2000
THE FAUCI FILES, Vol 3( 26): HIV Cocktail Warnings Ignored From
H.A.A.R.T. to F.A.A.R.T.
January 31, 2000
1996: H.A.A.R.T. = Highly Active Anti-Retroviral Therapy
2000: F.A.A.R.T. = Failed AIDS Accelerating Retroviral Torture
Under the tutelage of NIH/NIAID Dictator-Direktor Dr. Anthony
"Mussolini" Fauci, M.D., the HAART cocktails became the
"standard of care" in HIV treatment about four years ago.
Typical of Fauci's "leadership", these drug combinations
have neither been "standard" nor much in the way of "care",
and continued to be marketed with false claims despite
the explicit warnings published by leading AIDS researchers and
immunologists in regards to the harmful and immunosuppressive
nature of these drugs chosen for treating a disease of
Jay Levy warned:
"These observations strongly suggest that the immune system
has either been compromised by the drugs or 'put to rest'
by the therapy" (Lancet 1998; 352:982-83).
Giorgi's group warned:
"The most significant immunologic effect of combined protease
inhibitor and reverse transcriptase inhibitor therapy in our
study subjects was reversal of immune activation" (AIDS 1998,
Zinkernagel's group warned:
"Thus, the surprising ability of ritonavir to block the
presentation of antigen to CTLs may possibly contribute to
therapy of HIV infections but potentially also to the
therapy of virally induced immunopathology, autoimmune
diseases, and transplantation reactions" (PNAS 1998;
It has been "agreed," that CD8 CTLs are critical for
controlling HIV. It has also been shown that HAART
suppresses the various cytokines critical for activation
of CTLs and their homing/migration to the periphery
(AIDS 1998, 12:F123-F129; Journal of Infectious
Diseases 1997, 176:1175-1179).
Ullum et al. (AN Phillips was a co-author) warned:
"Low levels of IFN-gamma production were associated with an
increased risk of experiencing a CD4 count below 100 cells/m3
and death, analyzed in both univariate analysis and in
multivariate analysis adjusting for CD4 counts and age.
Thus, changes in production of IFN-gamma seem to be truly
related to the risk for disease progression in HIV infection"
(AIDS Res Hum Retroviruses 1997, 13:1039-46).
Andersson et al. (Landay's group) warned of HAART-induced suppression
"HIV-infected tonsil tissue was characterised by extensive
proinflammatory and type 1 cytokine expression. A five- to 15-fold
elevation of (IL)-1a, IL-12, IL-2, and interferon (IFN)-gamma
protein expression was found compared to controls, and each
encompassed a mean of at least 4.5% of the tissue compartment.
This was reduced by 20-90% in all individuals after 4 weeks of
HAART" (AIDS 1998, 12:F123-F129).
One might consider that you can't have it both ways. If IFN-
gamma is critical for survival and HAART suppresses/changes
production of IFN-gamma in the very important lymphoid
tissues, then it would be counterintuitive to believe that
HAART increases survival.
As shown by Levy, Giorgi and Zinkernagel, HAART is
suppressing the immune response thereby suppressing
proliferation of HIV-infected T cells. This has been
substantiated by the almost immediate rebound of plasma
viremia when HAART use is terminated.
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