THE FAUCI FILES, Vol 3(20): HIV Cocktail Fascist Myth vs. The Scientific Reality

fred fredshaw at primenet.com
Sat Jan 22 11:24:38 EST 2000


THE FAUCI FILES, Vol 3(20): HIV Cocktail Fascist Myth vs. The Scientific
Reality

January 20, 2000

The research cited below originated from an NIH/NIAID grant database.
NIH/NIAID Dr. Anthony "Mussolini" Fauci has once again subverted the
process of scientific discovery by his FAILURE to publish this
CRUCIAL data in the peer-reviewed scientific journals.

The reason why Fauci subverts the truth is obvious: this data
destroys the superstitious underlying premise that survival by
HIV-positive individuals depends on suppressing the viral load.

The Cocktail Fascist Myth:

  On Tue, 18 Jan 2000 09:36:37 -0800, in
  <180120000936370851%marnix at u.washington.edu> 
  Dr. Marnix Bosch, University of Washington, Seattle
  <marnix at u.washington.edu> wrote:

  "(We are using HAART to treat HIV infection) because it 
   suppresses HIV-1 replication, reduces the incidence of
   opportunistic infections and malignancies, and extends lifes."

The Scientific Reality:

THE FAUCI FILES, Vol 2(52): Long-Term Non-Progressors Have HIGH Levels
of Plasma Viremia !!!

13 Nov 1999 20:31:07 GMT


Here, in Fauci's own words, an admission that the viral load
lab test has been a drug marketing hoax from day #1:

   "Of interest was the fact that relatively high levels 
    of plasma viremia were noted in the majority of LTNPs, 
    comparable to levels in HIV-infected individuals 
    whose disease had progressed."

Fauci has known ALL along that the immune system is capable
of controlling HIV -- yet the HAART Hoax continued to shut
down the CD8/Cytotoxic T-Lymphocytes (CTLs) that WERE
ABSOLUTELY CRITICAL TO THE CONTROL OF HIV AND OPPORTUNISTIC
INFECTIONS !!! What's worse -- Fauci also presided over the
CD4+ T-cell count HOAX as the primary indicator of disease
progression -- in fact, the CD8/CTL cell count is a FAR
better predictor of HIV disease progression (people who have
ZERO CD4 cells can be asymptomatic -- but ONLY if their
CD8/CTL cell counts are relatively high (over 400-500).

Strangely enough, the LTNP "high viremia" information was 
not published in the biomedical journals -- it appeared in a 
NIAID-internal grant request and carried Toni "The Rat" 
Fauci's name as author (as always ... full article below).

Crooked Murdering Bastards!

fred

----------------------------------------------------------
Z01AI00675                           FAUCI, A S               

PROJECT NUMBER                       Z01 AI00675-03
TITLE STUDIES OF HIV INFECTED LONGTERM NON-PROGRESSORS

INSTITUTE AI                       FY  95    FAUCI, A S
INTRAMURAL UNITLIR                           NIAID, NIH
AWARD AMOUNT.........         $0
FUTURE YEARS

ABSTRACT:
 
We have studied a group (n=23) of HIV-infected individuals 
termed long-term non-progressors (LTNPs) whose disease has not 
progressed over periods ranging from 7 to 17 years.  Levels of 
viral burden and virus replication were quite low in peripheral 
blood (PB) and lymph node (LN) mononuclear cells (MC) of LTNPs 
compared to progressors.  Of interest was the fact that 
relatively high levels of plasma viremia were noted in the 
majority of LTNPs, comparable to levels in HIV-infected 
individuals whose disease had progressed.  The degree of 
follicular hyperplasia and the total nodal and germinal center 
areas were significantly less in LTNPs.  Of note, and in 
striking contrast to progressors, the lymph node architecture 
of the LTNPs were preserved despite several years of infection.  
Variable degrees of virus trapping was detected; several 
patients had little if any trapping of extracellular virions.  
Virus particles were virtually never detected in tissue or cell 
suspensions by electron microscopy.  Only rarely were 
individual cells detected that were expressing HIV.  The lack 
of virus trapping could in part explain the fact that although 
only very few cells were actively producing virus in LN, plasma 
viremia seemed not to be efficiently cleared leading to 
relatively high levels of plasma viremia.  These data together 
with studies of lymphoid tissue in progressors suggests that 
disease progression is at least in part related to the 
deposition of virions on the follicular dendritic cell network 
of LN germinal centers, persistent activation of LN, active 
virus replication, and progressive destruction of lymphoid 
tissue.  From an immunological standpoint, HIV-specific 
cytotoxicity against gag proteins was consistently observed in 
PBMC of LTNPs.  Proliferative responses to a variety of stimuli 
(mitogens, alloantigens, and recall antigens) were preserved in 
PBMC of LTNPs.  Characterization of humoral immune responses is 
currently underway.  Intensive study of LTNPs should shed 
valuable insight into the pathogenic mechanisms of HIV disease 
and hopefully will provide important information for the 
development of therapeutic and vaccine strategies.
 
CRISP INDEXING TERMS FROM CRISP THESAURUS:
09445965P   pathologic process
10357240P   host organism interaction
15606305P   HIV infection
30997957P   latent virus infection
04321437S   dendritic cell
04325673S   lymph node
04358925S   monocyte
30996564S   virus replication
40000238S   lymphocyte proliferation
40000359S   viremia
14652636T   human tissue
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