THE FAUCI FILES, Vol 3(20): HIV Cocktail Fascist Myth vs. The Scientific Reality
fredshaw at primenet.com
Sat Jan 22 11:24:38 EST 2000
THE FAUCI FILES, Vol 3(20): HIV Cocktail Fascist Myth vs. The Scientific
January 20, 2000
The research cited below originated from an NIH/NIAID grant database.
NIH/NIAID Dr. Anthony "Mussolini" Fauci has once again subverted the
process of scientific discovery by his FAILURE to publish this
CRUCIAL data in the peer-reviewed scientific journals.
The reason why Fauci subverts the truth is obvious: this data
destroys the superstitious underlying premise that survival by
HIV-positive individuals depends on suppressing the viral load.
The Cocktail Fascist Myth:
On Tue, 18 Jan 2000 09:36:37 -0800, in
<180120000936370851%marnix at u.washington.edu>
Dr. Marnix Bosch, University of Washington, Seattle
<marnix at u.washington.edu> wrote:
"(We are using HAART to treat HIV infection) because it
suppresses HIV-1 replication, reduces the incidence of
opportunistic infections and malignancies, and extends lifes."
The Scientific Reality:
THE FAUCI FILES, Vol 2(52): Long-Term Non-Progressors Have HIGH Levels
of Plasma Viremia !!!
13 Nov 1999 20:31:07 GMT
Here, in Fauci's own words, an admission that the viral load
lab test has been a drug marketing hoax from day #1:
"Of interest was the fact that relatively high levels
of plasma viremia were noted in the majority of LTNPs,
comparable to levels in HIV-infected individuals
whose disease had progressed."
Fauci has known ALL along that the immune system is capable
of controlling HIV -- yet the HAART Hoax continued to shut
down the CD8/Cytotoxic T-Lymphocytes (CTLs) that WERE
ABSOLUTELY CRITICAL TO THE CONTROL OF HIV AND OPPORTUNISTIC
INFECTIONS !!! What's worse -- Fauci also presided over the
CD4+ T-cell count HOAX as the primary indicator of disease
progression -- in fact, the CD8/CTL cell count is a FAR
better predictor of HIV disease progression (people who have
ZERO CD4 cells can be asymptomatic -- but ONLY if their
CD8/CTL cell counts are relatively high (over 400-500).
Strangely enough, the LTNP "high viremia" information was
not published in the biomedical journals -- it appeared in a
NIAID-internal grant request and carried Toni "The Rat"
Fauci's name as author (as always ... full article below).
Crooked Murdering Bastards!
Z01AI00675 FAUCI, A S
PROJECT NUMBER Z01 AI00675-03
TITLE STUDIES OF HIV INFECTED LONGTERM NON-PROGRESSORS
INSTITUTE AI FY 95 FAUCI, A S
INTRAMURAL UNITLIR NIAID, NIH
AWARD AMOUNT......... $0
We have studied a group (n=23) of HIV-infected individuals
termed long-term non-progressors (LTNPs) whose disease has not
progressed over periods ranging from 7 to 17 years. Levels of
viral burden and virus replication were quite low in peripheral
blood (PB) and lymph node (LN) mononuclear cells (MC) of LTNPs
compared to progressors. Of interest was the fact that
relatively high levels of plasma viremia were noted in the
majority of LTNPs, comparable to levels in HIV-infected
individuals whose disease had progressed. The degree of
follicular hyperplasia and the total nodal and germinal center
areas were significantly less in LTNPs. Of note, and in
striking contrast to progressors, the lymph node architecture
of the LTNPs were preserved despite several years of infection.
Variable degrees of virus trapping was detected; several
patients had little if any trapping of extracellular virions.
Virus particles were virtually never detected in tissue or cell
suspensions by electron microscopy. Only rarely were
individual cells detected that were expressing HIV. The lack
of virus trapping could in part explain the fact that although
only very few cells were actively producing virus in LN, plasma
viremia seemed not to be efficiently cleared leading to
relatively high levels of plasma viremia. These data together
with studies of lymphoid tissue in progressors suggests that
disease progression is at least in part related to the
deposition of virions on the follicular dendritic cell network
of LN germinal centers, persistent activation of LN, active
virus replication, and progressive destruction of lymphoid
tissue. From an immunological standpoint, HIV-specific
cytotoxicity against gag proteins was consistently observed in
PBMC of LTNPs. Proliferative responses to a variety of stimuli
(mitogens, alloantigens, and recall antigens) were preserved in
PBMC of LTNPs. Characterization of humoral immune responses is
currently underway. Intensive study of LTNPs should shed
valuable insight into the pathogenic mechanisms of HIV disease
and hopefully will provide important information for the
development of therapeutic and vaccine strategies.
CRISP INDEXING TERMS FROM CRISP THESAURUS:
09445965P pathologic process
10357240P host organism interaction
15606305P HIV infection
30997957P latent virus infection
04321437S dendritic cell
04325673S lymph node
30996564S virus replication
40000238S lymphocyte proliferation
14652636T human tissue
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