In article <3en9d8$118a at ns1.CC.Lehigh.EDU>, <x011 at ns1.CC.Lehigh.EDU> wrote:
>In article <ewass-0601950950080001 at hmc25.ninds.nih.gov>,
>ewass at helix.nih.gov (Eric Wassermann) writes:
>>In article <D1yKGu.Au8 at murdoch.acc.Virginia.EDU>,
>>mlm2s at galen.med.Virginia.EDU (Mallory Leslie McClure) wrote:
>>>>> How is it possible to have a patient have a stroke....
>>I had hoped someone else would post this because I am doing it
>from memory. The active chemical in cough syrup at very high
>doses will stop the nerves from killing themselves after damage.
>One may wait up to 24 hours to give the medicine.
>Ron Blue
Let's not jump the gun here. Dextromethorphan and other
excitatory amino acid antagonists are in clinical trials
for stroke treatment, but at present there are no human
data to show that any of them work. There are many other
approaches being tried (thrombolysis, free radical scavengers,
other channel blockers, etc.); hopefully one or more
will emerge as useful treatments, but none has as of yet.
Regarding the original question (how could the findings of
the stoke be gone some months later), there are many
possible explanations. In addition to the ones already
suggested, there is a great deal about the reorganization
of brain function after injury that we don't understand
mechanistically, but which seems to indicate that other
pathways and gray matter structures assume the 'missing'
functions.
--
Tom Bleck (Thomas P. Bleck, M.D.) tbleck at virginia.edu
Departments of Neurology and Neurological Surgery
University of Virginia School of Medicine
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