I think the underlying question would be what it causing the bifrontal
slowing., hence the recommendation of brain imaging. Frontal slowing is
often accompanied by difficulties with executive functioning which could
include as difficulties in planning, organization, following long-term
goals, making good decisions, abstract thinking etc. It could also manifest
as changes in personality such as impusivity, inertia, changes in social
conduct, mood changes, and irritability. If you experience any of these, it
might be a good idea to see a neuropsychologist who could test you more
formally and explain your symptoms in terms of the medical findings.
MailCA <mailca at aol.com> wrote in message
news:20010105172344.04556.00000080 at ng-mi1.aol.com...
> The following is an EEG report from about a year and a half ago. I was
> diagnosed with narcolepsy.
>> I'm not well-versed in neurosciences, but I know that narcolepsy is
> an intrusion of REM brain patterns into normal waking time. And from this
> report, it appears that there is an abnormal amount of slow wave activity
> I'm awake-- kind of like my brain is partially asleep.
>> My question is whether this EEG should have suggested further neurological
> inquiry (besides just the CAT scan they had me in for), which might have
> to the narcolepsy diagnosis earlier. I would also appreciate any input
> other possible implications from this report.
> Christy Ann
> A 21 channel recording with standard 10-20 electrode placements after
> hydrate desation. Hyperventilation and photic stimulation were performed.
> During wakefulness there is moderate amplitude 10 Hz rhythmic posterior
> activity which is reactive to eye opening. Throughout the study there is
> bifrontal independent polymorphic delta activity to varying extents. Some
> this seems to be related to eye motion artifact but other appears to be of
> cerebral origin. No paroxysmal or epileptiform activity is noted and no
> of sleep are achieved. During drowsiness generalized irregular low
> slow waves predominate. Hyperventilation leads to slight build-up of theta
> activity without focality. Photic stimulation elicits no changes.
> This is a mildly abnormal EEG because of excess bifrontal irregular slow
> activity. Some of this is likely to be of cerebral origin and could
> area of localized cortical dysfunction which could be on a structural,
> traumatic, ischemic, or post ictal basis. Brain imaging may be advisable.