[Neuroscience] Re: Carotenoid Transport into the RPE

John H. via neur-sci%40net.bio.net (by johnh from goawayplease.com)
Tue Dec 11 06:25:08 EST 2007


We just received a bucket of studies from the principle researcher in the 
Blue Mountains study. Her suggestion, and I tend to agree with it, is that 
zinc is protective. Understand that drusen and lipofuscin contains all sorts 
of things, even amyloid. The problem is failure of phagocytosis via cd 36 at 
the RPE and additionally insufficient lysosomal degradation. Keep the focus 
on Lutein and zeaxanthin.


"Don W" <dwilgus from prodigy.net> wrote in message 
news:JMh7j.29740$lD6.4694 from newssvr27.news.prodigy.net...
> John,
>  Noticed (after I sent the last note) that a/the LAST study (Lutein 
> antioxidant supplementation 2004 study) also tied in macular pigment 
> increases with lutein intake _and_ the acuity increases.  And just 
> recently (as far as my (this early am) searches) there is a LAST II study 
> report in Optometry (May 2007) by Richer that discusses macular pigment 
> increases (please see PubMed if interested).  To me, this is most 
> profound, take is to take a supplement and to see (and measure (several 
> techniques available, also!!)) the result.  And hopefully, have possible 
> acuity increases in this process.  Will try to get the full Aleman's paper 
> and Richer's paper. Abstracts leave out too much.
>  So where are we with what you think the zinc level should be?
>  Re wired implants:  There is something bothersome to see a neuron axon 
> (dendrite) draped across a silicon substrate tied to a terminal post. 
> Wireless anyone?
>  Other stuff .... later.
> Don W.

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